Document Detail


Detection of JC virus DNA sequences in colorectal cancers in Japan.
MedLine Citation:
PMID:  16021515     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
JC virus (JCV), a ubiquitous polyoma virus that commonly infects humans, was first identified as the etiologic agent for the fetal demyelinating disease, progressive multifocal leukoencephalopathy. Recently, a number of reports have documented detection of JCV in samples derived from several types of neural as well as non-neural human tumors. It has been suggested that oncogenicity of JCV depends on a T antigen having a strict structural homology to the T antigen of simian virus 40. To clarify whether JCV might have a potential role with regard to colorectal cancers, we investigated the presence of its genome in a series of cases along with colorectal adenomas and normal colonic mucosa, targeting T antigen, VP and agnoprotein by nested polymerase chain reaction and Southern blotting and T antigen by immunohistochemistry. While VP and agnoprotein were not found in any of the samples examined, T antigen was detected in 6 of 23 colorectal cancers (26.1%) and 1 of 21 adenomas (4.8%), but none of 20 samples of normal colonic mucosa. No clear and diffuse staining with anti-T-antigen antibodies (1:100) could be detected, and there was no correlation with CD20-positive cells, which might have indicated JCV latent infection of B lymphocytes. Presence of T antigen did not influence clinicopathological variables, including survival. In one colonic cancer case positive for T antigen together with lymph node metastasis, DNA extracted from cancer cells in the lymph node revealed no detection of T antigen. Our results are in the intermediate position between the high T antigen rate (81%) in one report and the lack of it (0%) in another focused on colon cancers. It was concluded that T antigen might be integrated in cancer cells in approximately one fourth of Japanese colon cancer cases without clear and diffuse expression of the protein, suggesting a possible role in oncogenesis which might involve a hit-and-run mechanism.
Authors:
Ryouta Hori; Yoshihiro Murai; Kouichi Tsuneyama; Hekmat Osman Abdel-Aziz; Kazuhiro Nomoto; Hiroyuki Takahashi; Chun-mei Cheng; Tomohiko Kuchina; Brian V Harman; Yasuo Takano
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Publication Detail:
Type:  Comparative Study; Journal Article     Date:  2005-10-19
Journal Detail:
Title:  Virchows Archiv : an international journal of pathology     Volume:  447     ISSN:  0945-6317     ISO Abbreviation:  Virchows Arch.     Publication Date:  2005 Oct 
Date Detail:
Created Date:  2005-11-02     Completed Date:  2006-01-18     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  9423843     Medline TA:  Virchows Arch     Country:  Germany    
Other Details:
Languages:  eng     Pagination:  723-30     Citation Subset:  IM    
Affiliation:
First Department of Pathology, Faculty of Medicine, Toyama Medical and Pharmaceutical University, 2630 Sugitani, Toyama, 930-0152, Japan, ytakano@ms.toyama-mpu.ac.jp.
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MeSH Terms
Descriptor/Qualifier:
Adenocarcinoma / pathology,  virology*
Adenoma / pathology,  virology*
Adolescent
Aged
Aged, 80 and over
Antigens, CD20 / metabolism
Antigens, Viral, Tumor / analysis
Blotting, Southern
Colorectal Neoplasms / pathology,  virology*
DNA, Viral / analysis*
Female
Humans
Immunohistochemistry
JC Virus / genetics*,  immunology
Japan
Male
Middle Aged
Polymerase Chain Reaction
Prognosis
Tumor Virus Infections / epidemiology*
Viral Proteins / analysis
Viral Regulatory and Accessory Proteins
Chemical
Reg. No./Substance:
0/Antigens, CD20; 0/Antigens, Viral, Tumor; 0/DNA, Viral; 0/Viral Proteins; 0/Viral Regulatory and Accessory Proteins; 0/agnoprotein, polyomavirus
Comments/Corrections
Comment In:
Virchows Arch. 2006 Feb;448(2):239   [PMID:  16365728 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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