Document Detail


Detection of CHD7 deletions by MLPA in CHARGE syndrome patients with a less typical phenotype.
MedLine Citation:
PMID:  19248844     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Bergman et al. performed a search for exon copy number alterations in the CHD7 gene using MLPA in CHARGE syndrome patients who did not have a CHD7 mutation. Based on their results they recommended to extend testing using MLPA solely in individuals with a typical CHARGE syndrome phenotype. However, since we have found deletions comprising the CHD7 gene in three patients with a less typical phenotype we recommend performing MLPA testing in all CHARGE syndrome patients without causal CHD7 mutations.
Authors:
Josephine Wincent; Astrid Schulze; Jacqueline Schoumans
Related Documents :
20865644 - New treatment options for atypical hemolytic uremic syndrome with the complement inhibi...
11039354 - Prominent basal emissary foramina in syndromic craniosynostosis: correlation with pheno...
1920914 - Refined determination of breakpoints of the translocation t(1;7) associated with signs ...
20949524 - Natural history of christianson syndrome.
20002364 - The value of lung ultrasound monitoring in h1n1 acute respiratory distress syndrome.
2947784 - Correlation of absent inner dynein arms and mucociliary clearance in a patient with kar...
Publication Detail:
Type:  Comment; Letter     Date:  2009-02-25
Journal Detail:
Title:  European journal of medical genetics     Volume:  52     ISSN:  1878-0849     ISO Abbreviation:  Eur J Med Genet     Publication Date:    2009 Jul-Aug
Date Detail:
Created Date:  2009-07-27     Completed Date:  2009-11-02     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101247089     Medline TA:  Eur J Med Genet     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  271-2     Citation Subset:  IM    
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Coloboma / genetics*
DNA Helicases / genetics*
DNA Mutational Analysis*
DNA-Binding Proteins / genetics*
Ear / abnormalities*
Exons
Heart Defects, Congenital / genetics*
Humans
Mental Retardation / genetics*
Nucleic Acid Amplification Techniques*
Phenotype
Sequence Deletion / genetics*
Syndrome
Chemical
Reg. No./Substance:
0/DNA-Binding Proteins; EC 3.6.1.-/DNA Helicases; EC 5.99.-/CHD7 protein, human
Comments/Corrections
Comment On:
Eur J Med Genet. 2008 Sep-Oct;51(5):417-25   [PMID:  18472328 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Immunomodulatory role of prolactin in diabetes development.
Next Document:  Fruit and vegetable consumption and its relation to markers of inflammation and oxidative stress in ...