| Destruction of elastic tissue (elastolysis) as a link between atherosclerosis and the temporal arteritis/polymyalgia rheumatica syndrome. Observations on an actinic factor in vascular disease. | |
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MedLine Citation:
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PMID: 6366708 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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Destruction (elastolysis) of the internal elastic lamina is frequently observed near early atherosclerotic plaques. Elastolysis and plaque formation are also found together in the temporal arteritis/polymyalgia rheumatica syndrome. Could it be that atherosclerosis and the syndrome are more closely akin than usually thought, with elastolysis acting as the pathogenetic link between them? A kinship of this nature is in accord with the growing recognition that elastin-related autoimmunity prevails in both these forms of vascular disease. A case can also be made out for the belief that the autoimmune reactions in internal vessels may be provoked by events in the integument where a slow but ultimately massive turnover of dermal and vascular elastic tissue takes place under the harmful influence of solar and other forms of actinic radiation. |
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Authors:
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J P O'Brien |
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Publication Detail:
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Type: Journal Article; Review |
Journal Detail:
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Title: Pathologie-biologie Volume: 32 ISSN: 0369-8114 ISO Abbreviation: Pathol. Biol. Publication Date: 1984 Feb |
Date Detail:
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Created Date: 1984-04-12 Completed Date: 1984-04-12 Revised Date: 2007-11-15 |
Medline Journal Info:
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Nlm Unique ID: 0265365 Medline TA: Pathol Biol (Paris) Country: FRANCE |
Other Details:
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Languages: eng Pagination: 123-38 Citation Subset: IM |
Export Citation:
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| MeSH Terms | |
Descriptor/Qualifier:
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Arteriosclerosis
/
immunology,
metabolism*,
pathology Elastic Tissue / immunology, metabolism*, pathology Giant Cell Arteritis / immunology, metabolism*, pathology Humans Light Polymyalgia Rheumatica / immunology, metabolism*, pathology |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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