Document Detail


Depletion of Ly6G/C(+) cells ameliorates delayed cerebral vasospasm in subarachnoid hemorrhage.
MedLine Citation:
PMID:  21059474     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: The etiology of delayed cerebral vasospasm (DCV) after aneurysmal subarachnoid hemorrhage (SAH) has remained elusive. Growing evidence supports a role for inflammation in the pathogenesis of DCV. We showed that CSF neutrophils predict which patients will develop DCV.
METHODS: We evaluated a murine model of SAH to test the hypothesis that myeloid cells are required for the cerebral damage associated with DCV.
RESULTS: SAH was associated with decreased middle cerebral artery caliber on day 1 which normalized at day 3 and recurred at day 6. In addition, behavioral testing with a Barnes maze showed executive dysfunction that progressively worsened after the seventh day post hemorrhage. To test the role of innate immune responses, we administrated a myeloid cell-depleting monoclonal antibody against Ly6G/C prior to experimental SAH. Myeloid cell depletion ameliorated angiographic vasospasm measured by MCA vessel caliber and normalized behavioral testing.
CONCLUSION: Our findings support the role of Ly6G/C(+) cells in the development of DCV after SAH and suggest that immune modulation of neutrophils or other Ly6G/C(+) cells may be a strategy for the prevention of DCV.
Authors:
J Javier Provencio; Tamer Altay; Saksith Smithason; Shari Korday Moore; Richard M Ransohoff
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2010-11-06
Journal Detail:
Title:  Journal of neuroimmunology     Volume:  232     ISSN:  1872-8421     ISO Abbreviation:  J. Neuroimmunol.     Publication Date:  2011 Mar 
Date Detail:
Created Date:  2011-03-09     Completed Date:  2011-05-17     Revised Date:  2012-03-07    
Medline Journal Info:
Nlm Unique ID:  8109498     Medline TA:  J Neuroimmunol     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  94-100     Citation Subset:  IM    
Copyright Information:
Copyright © 2010 Elsevier B.V. All rights reserved.
Affiliation:
Neuroinflammation Research Center, Lerner Research Institute, NB3, Cleveland Clinic, 9500 Euclid Ave. Cleveland, OH 44195, United States. provenj@ccf.org
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MeSH Terms
Descriptor/Qualifier:
Animals
Antigens, Ly / immunology,  metabolism
Cell Separation
Disease Models, Animal
Flow Cytometry
Immunohistochemistry
Inflammation / complications*,  immunology*
Male
Maze Learning
Mice
Mice, Inbred C57BL
Middle Cerebral Artery / immunology,  pathology
Myeloid Cells / immunology,  metabolism
Neutrophils / immunology*,  metabolism
Subarachnoid Hemorrhage / complications*,  immunology*,  pathology
Vasospasm, Intracranial / immunology*,  pathology
Grant Support
ID/Acronym/Agency:
K08 NS051350/NS/NINDS NIH HHS; K08 NS051350-04/NS/NINDS NIH HHS; K24 NS51400/NS/NINDS NIH HHS
Chemical
Reg. No./Substance:
0/Antigens, Ly; 0/Ly-6C antigen, mouse; 0/Ly6G antigen, mouse

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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