Document Detail


Delta9-tetrahydrocannabinol increases C6 glioma cell death produced by oxidative stress.
MedLine Citation:
PMID:  15975726     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
(-)Delta9-tetrahydrocannabinol is a scavenger of free radicals. However, the activation of the CB1 receptor in cultured C6 glioma cells by (-)delta9-tetrahydrocannabinol in the presence of reagents generating reactive oxygen species leads to amplification of the cellular damage from oxidative stress. This was evident by increased loss of cell wall integrity, impaired mitochondrial function and reduction of glucose uptake. In addition, (-)delta9-tetrahydrocannabinol treatment was also found to be deleterious to the cells under conditions of glucose starvation. Free radicals have been implicated in various conditions leading to cell death and, as a routine, the Fenton reaction is utilized for modeling reactive oxygen species production. Our study was performed using a cell permeating Fe(III) chelating quinone that provides more physiological conditions for mimicking the naturally occurring oxidative stress within the cell and thus serves as a better model for natural reactive oxygen species formation.
Authors:
I Goncharov; L Weiner; Z Vogel
Related Documents :
2690676 - A device for aeration and mixing of cell and organelle suspensions during nuclear magne...
18588206 - Diosgenin formation by freely suspended and entrapped plant cell cultures of dioscorea ...
2540066 - The cellular-induced decay of dmpo spin adducts of .oh and .o2.
9027726 - Immunohistochemical colocalization of the alpha-subunit of neutrophil nadph oxidase and...
2246326 - Inhibition of gap-junctional intercellular communication and enhanced binding of fibron...
21093146 - Coleusin factor exerts cytotoxic activity by inducing g0/g1 cell cycle arrest and apopt...
Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Neuroscience     Volume:  134     ISSN:  0306-4522     ISO Abbreviation:  Neuroscience     Publication Date:  2005  
Date Detail:
Created Date:  2005-08-08     Completed Date:  2005-11-15     Revised Date:  2005-11-17    
Medline Journal Info:
Nlm Unique ID:  7605074     Medline TA:  Neuroscience     Country:  United States    
Other Details:
Languages:  eng     Pagination:  567-74     Citation Subset:  IM    
Affiliation:
Department of Neurobiology, Weizmann Institute of Science, Herzel Street, Rehovot 76100, Israel. igor.goncharov@weizmann.ac.il
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Biological Transport
CHO Cells
Cell Death / drug effects*
Cell Line, Tumor
Cell Survival / drug effects
Cricetinae
Deoxyglucose / pharmacokinetics
Glioma / pathology*
Hela Cells
Humans
Oxidative Stress / physiology*
Receptor, Cannabinoid, CB1 / drug effects,  physiology
Tetrahydrocannabinol / toxicity*
Chemical
Reg. No./Substance:
0/Receptor, Cannabinoid, CB1; 154-17-6/Deoxyglucose; 1972-08-3/Tetrahydrocannabinol

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Dynamic response properties of visual neurons and context-dependent surround effects on receptive fi...
Next Document:  Autonomic brainstem nuclei are linked to the hippocampus.