Document Detail


Dehydroepiandrosterone pretreatment protects rats against the pro-oxidant and necrogenic effects of carbon tetrachloride.
MedLine Citation:
PMID:  8250954     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
A single intraperitoneal injection of dehydroepiandrosterone (3 beta-hydroxy-5-androsten-17-one, DHEA) 17 hr before carbon tetrachloride (CCl4) poisoning protects rats against liver injury induced by the haloalkane. In liver homogenates, both the increase in malondialdehyde production and the formation of fluorescent lipid peroxidation products are significantly reduced. Also, liver microsomes obtained from DHEA-pretreated rats incubated in vitro with CCl4 are less susceptible to lipid peroxidation than microsomes from normal animals. The release of liver enzymes into the blood is much reduced in DHEA-pretreated rats, confirming a cause-effect relationship between lipid peroxidation and hepatocyte death. Treatment with DHEA inhibits neither glucose-6-phosphate dehydrogenase activity in the cytosol, nor the microsomal mixed function oxidase system (cytochrome P450 content, aminopyrine demethylase and ethoxycoumarin de-ethylase activities). In animals treated with DHEA, the liver content of total glutathione and vitamin E is not modified. These results support the hypothesis that DHEA protects against CCl4-induced liver injury through its own antioxidant activity, rather than by interfering with the metabolism of the toxin or with the tissue level of primary antioxidants.
Authors:
M Aragno; E Tamagno; G Boccuzzi; E Brignardello; E Chiarpotto; A Pizzini; O Danni
Related Documents :
3342874 - A new pathway for phosphatidylserine synthesis in rat liver microsomes.
16720684 - N-glucuronidation of perfluorooctanesulfonamide by human, rat, dog, and monkey liver mi...
3801044 - Inhibitory effect of female hormones on lipid peroxidation.
3839164 - Monooxygenase induction by various xenobiotics and its influence on the rat liver micro...
9013444 - Reduced renal medullary water channel expression in puromycin aminonucleoside--induced ...
6508734 - The discovery of a rapidly metabolized polymeric tetraphosphate derivative of adenosine...
Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Biochemical pharmacology     Volume:  46     ISSN:  0006-2952     ISO Abbreviation:  Biochem. Pharmacol.     Publication Date:  1993 Nov 
Date Detail:
Created Date:  1994-01-04     Completed Date:  1994-01-04     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0101032     Medline TA:  Biochem Pharmacol     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  1689-94     Citation Subset:  IM    
Affiliation:
General Pathology Institute, Sassari, Italy.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Antioxidants / pharmacology*
Carbon Tetrachloride / antagonists & inhibitors*
Carbon Tetrachloride Poisoning / enzymology,  prevention & control*
Dehydroepiandrosterone / pharmacology*
Lipid Peroxidation / drug effects
Malondialdehyde / analysis
Microsomes, Liver / drug effects,  enzymology
Rats
Chemical
Reg. No./Substance:
0/Antioxidants; 53-43-0/Dehydroepiandrosterone; 542-78-9/Malondialdehyde; 56-23-5/Carbon Tetrachloride

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Comparison of hamster and mouse reveals interspecies differences in the regulation of hepatic CYP2A ...
Next Document:  Hydroxyl radical damage to DNA sugar and model membranes induced by anthralin (dithranol).