Document Detail


Decreased apolipoprotein A5 is implicated in insulin resistance-related hypertriglyceridemia in obesity.
MedLine Citation:
PMID:  20047745     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Hypertriglyceridemia is associated insulin resistance in obese people. Recently identified apolipoprotein A5 (apoA5) is involved in triglyceride (TG) metabolism. This study was to investigate the role of apoA5 in insulin resistance-related hypertriglyceridemia in obesity. 682 participants including 340 non-obese individuals and 342 obese individuals were recruited in this study. Plasma apoA5 levels were measured. The insulin resistance in participants was assessed by homeostasis model assessment of insulin resistance (HOMA-IR). An insulin resistant and hypertriglyceridemic rat model was established by high-fructose diet with obese Zucker rats as positive controls. Besides, two insulin resistant models in vitro were induced by insulin and tumor necrosis factor-alpha (TNFalpha) in HepG2 cells. Obese participants had lower plasma apoA5 levels. Plasma apoA5 levels were inversely correlated with TG, body mass index and HOMA-IR in humans. Furthermore, hepatic and plasma apoA5 reduced in fructose-fed rats whereas no significant changes of apoA5 were observed in obese Zucker rats. In addition, treatment of HepG2 cells with insulin and TNFalpha decreased apoA5 expression and increased TG content. Thus, decreased apolipoprotein A5 is implicated in insulin resistance-related hypertriglyceridemia in obesity.
Authors:
Xian-sheng Huang; Shui-Ping Zhao; Min Hu; Lin Bai; Qian Zhang; Wang Zhao
Related Documents :
16208285 - Relationship between percentage of vo2max and type of physical activity in obese and no...
16448525 - Prediction of response to sibutramine therapy in obese non-diabetic and diabetic patients.
2036185 - Inner ear damage secondary to diabetes mellitus. i. changes in adolescent shr/n-cp rats.
18537725 - Development of 11beta-hsd1 inhibitors for the treatment of type 2 diabetes.
21388025 - Cases on the rise: current diagnosis guidelines and research efforts for a cure.
16481885 - From big fat cells to high blood pressure: a pathway to obesity-associated hypertension.
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2009-12-11
Journal Detail:
Title:  Atherosclerosis     Volume:  210     ISSN:  1879-1484     ISO Abbreviation:  Atherosclerosis     Publication Date:  2010 Jun 
Date Detail:
Created Date:  2010-05-31     Completed Date:  2010-12-06     Revised Date:  2011-05-06    
Medline Journal Info:
Nlm Unique ID:  0242543     Medline TA:  Atherosclerosis     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  563-8     Citation Subset:  IM    
Copyright Information:
Copyright (c) 2010 Elsevier Ireland Ltd. All rights reserved.
Affiliation:
Department of Cardiology, The Second Xiang Ya Hospital, Central South University, No. 139, Renmin Zhonglu, Changsha, Hunan, 410011, China.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Adult
Animals
Apolipoproteins / blood*
Apolipoproteins A / blood*
Female
Glucose Clamp Technique
Humans
Hypertriglyceridemia / complications*
Insulin / metabolism
Insulin Resistance*
Male
Middle Aged
Obesity / complications*
Rats
Rats, Zucker
Tumor Necrosis Factor-alpha / metabolism
Chemical
Reg. No./Substance:
0/APOA5 protein, human; 0/Apoa5 protein, rat; 0/Apolipoproteins; 0/Apolipoproteins A; 0/Tumor Necrosis Factor-alpha; 11061-68-0/Insulin
Comments/Corrections
Comment In:
Atherosclerosis. 2010 Oct;212(2):386-7   [PMID:  20638662 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Ala54Thr polymorphism of fatty acid-binding protein 2 gene and fasting blood lipids: a meta-analysis...
Next Document:  Assessment of amputee socket-stump-residual bone kinematics during strenuous activities using Dynami...