Document Detail


Daclizumab: a potential asthma therapy?
MedLine Citation:
PMID:  20804450     Owner:  NLM     Status:  In-Process    
Abstract/OtherAbstract:
Airways inflammation in asthma is triggered and maintained by CD4+ (Th2) cells which are activated by IL-2 and stimulate the eosinophilic inflammation, IgE secretion and mucus hyperproduction. Current anti-inflammatory therapies include inhaled corticosteroids, and leukotriene modifiers but they are not universally effective. IL-2 pathway inhibition might represent a potent anti-inflammatory therapy in asthma given the cytokine early role in the asthma complex inflammatory cascade and daclizumab which is a IL-2R blocker currently used as an immunsupressor in organ transplantation might be a potential asthma therapy. This is review on the pathogenic role of IL-2, on the therapeutic potential of daclizumab and on its related patents.
Authors:
Sabina A Antoniu
Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Recent patents on inflammation & allergy drug discovery     Volume:  4     ISSN:  1872-213X     ISO Abbreviation:  Recent Pat Inflamm Allergy Drug Discov     Publication Date:  2010 Nov 
Date Detail:
Created Date:  2010-09-30     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101309297     Medline TA:  Recent Pat Inflamm Allergy Drug Discov     Country:  United Arab Emirates    
Other Details:
Languages:  eng     Pagination:  214-21     Citation Subset:  IM    
Affiliation:
Gr T Popa University of Medicine and Pharmacy Iaşi, Department of Medicine II -Pulmonary Disease, Pulmonary University Hospital, 30 Dr I Cihac Str, 700115 Iasi, Romania. sabina.antonela.antoniu@pneum.umfiasi.ro
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Antagonism of eosinophil accumulation in asthma.
Next Document:  Review article: stem cell therapies for inflammatory bowel disease - efficacy and safety.