Document Detail


DNA replication checkpoint promotes G1-S transcription by inactivating the MBF repressor Nrm1.
MedLine Citation:
PMID:  18682565     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The cell cycle transcriptional program imposes order on events of the cell-cycle and is a target for signals that regulate cell-cycle progression, including checkpoints required to maintain genome integrity. Neither the mechanism nor functional significance of checkpoint regulation of the cell-cycle transcription program are established. We show that Nrm1, an MBF-specific transcriptional repressor acting at the transition from G(1) to S phase of the cell cycle, is at the nexus between the cell cycle transcriptional program and the DNA replication checkpoint in fission yeast. Phosphorylation of Nrm1 by the Cds1 (Chk2) checkpoint protein kinase, which is activated in response to DNA replication stress, promotes its dissociation from the MBF transcription factor. This leads to the expression of genes encoding components that function in DNA replication and repair pathways important for cell survival in response to arrested DNA replication.
Authors:
R A M de Bruin; T I Kalashnikova; A Aslanian; J Wohlschlegel; C Chahwan; J R Yates; P Russell; C Wittenberg
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2008-08-05
Journal Detail:
Title:  Proceedings of the National Academy of Sciences of the United States of America     Volume:  105     ISSN:  1091-6490     ISO Abbreviation:  Proc. Natl. Acad. Sci. U.S.A.     Publication Date:  2008 Aug 
Date Detail:
Created Date:  2008-08-13     Completed Date:  2008-09-16     Revised Date:  2011-11-02    
Medline Journal Info:
Nlm Unique ID:  7505876     Medline TA:  Proc Natl Acad Sci U S A     Country:  United States    
Other Details:
Languages:  eng     Pagination:  11230-5     Citation Subset:  IM    
Affiliation:
Departments of Molecular Biology and Cell Biology, The Scripps Research Institute, 10550 North Torrey Pines Road, La Jolla, CA 92037, USA.
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MeSH Terms
Descriptor/Qualifier:
DNA Repair / physiology
DNA Replication / physiology*
DNA, Fungal / genetics,  metabolism*
G1 Phase / physiology*
Genome, Fungal / physiology
Phosphorylation
Protein-Serine-Threonine Kinases / genetics,  metabolism
Repressor Proteins / genetics,  metabolism*
S Phase / physiology*
Schizosaccharomyces / genetics,  metabolism*
Schizosaccharomyces pombe Proteins / genetics,  metabolism*
Grant Support
ID/Acronym/Agency:
CA77325/CA/NCI NIH HHS; GM059441/GM/NIGMS NIH HHS; GM59447/GM/NIGMS NIH HHS
Chemical
Reg. No./Substance:
0/DNA, Fungal; 0/Nrm1 protein, S pombe; 0/Repressor Proteins; 0/Schizosaccharomyces pombe Proteins; EC 2.7.1.11/checkpoint kinase 2; EC 2.7.11.1/Protein-Serine-Threonine Kinases
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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