Document Detail


DNA methylation: its contribution to systemic lupus erythematosus.
MedLine Citation:
PMID:  17061057     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Recent studies on epigenetics, including the methylation of DNA and the enzymes regulating methylation, seem likely to contribute to understanding the pathogenesis of autoimmune diseases such as systemic lupus erythematosus (SLE). In fact, the relationship between DNA methylation and SLE has long been the subject of investigation. To obtain a deeper understanding of the role of DNA methylation in the onset of SLE, we reviewed the findings reported in the literature and our own data about DNA methylation and SLE. Various studies have indicated the possible importance of DNA methylation, especially hypomethylation, in the aetiology of SLE. Epigenetic studies may provide clues for elucidating the pathogenesis of SLE and for developing new strategies to treat this disease.
Authors:
I Sekigawa; M Kawasaki; H Ogasawara; K Kaneda; H Kaneko; Y Takasaki; H Ogawa
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Clinical and experimental medicine     Volume:  6     ISSN:  1591-8890     ISO Abbreviation:  Clin. Exp. Med.     Publication Date:  2006 Oct 
Date Detail:
Created Date:  2006-10-24     Completed Date:  2007-01-03     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  100973405     Medline TA:  Clin Exp Med     Country:  Italy    
Other Details:
Languages:  eng     Pagination:  99-106     Citation Subset:  IM    
Affiliation:
Department of Internal Medicine, Juntendo University Urayasu Hospital, 2-1-1 Tomioka, Urayasu-shi, Chiba, 279-0021, Japan. sekigawa@juntendo-urayasu.jp
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MeSH Terms
Descriptor/Qualifier:
Animals
CpG Islands
DNA Methylation*
Endogenous Retroviruses / genetics
Epigenesis, Genetic
Humans
Lupus Erythematosus, Systemic / etiology,  genetics*,  metabolism*
Mice
Toll-Like Receptors / immunology
Transcription, Genetic
Chemical
Reg. No./Substance:
0/Toll-Like Receptors

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