Document Detail


Cytokines and cytokine receptors in acute lymphoblastic leukemia expressing myeloid markers--role in growth regulation.
MedLine Citation:
PMID:  7873998     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
There is no evidence that cancer cells including leukemic cells are immortal. It has been clearly indicated that certain cytokines can significantly stimulate leukemic cell proliferation in vitro, and sustain the circuit of autocrine or paracrine stimulation. The biological roles of cytokines and cytokine receptors have been intensively investigated in acute leukemia. Recently coexpression of both lymphoid and myeloid features on a single leukemic cell has been well recognized using a flowcytometric technique. Studies of ALL cells expressing myeloid markers (My+ ALL) have indicated that the profiles of cytokines and cytokine receptors expressed by My+ ALL show both similarities and differences to those in My- ALL or acute myelogenous leukemia (AML), suggesting that My+ ALL cells may originate from uncommitted hematopoietic precursor cells coexpressing features of both lymphoid and myeloid lineages. The exact assessment of cytokine response of leukemic cells would provide an important tool for phenotyping acute leukemia based on the growth properties of the cells (cytokine phenotyping), in addition to the morphologic classification and immunological surface phenotyping. Additionally alteration of sensitivity to cytotoxic anticancer drugs by cytokine stimulation may be the new strategy for biologic therapy of acute leukemia.
Authors:
Y Komada; M Sakurai
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Leukemia & lymphoma     Volume:  15     ISSN:  1042-8194     ISO Abbreviation:  Leuk. Lymphoma     Publication Date:  1994 Nov 
Date Detail:
Created Date:  1995-04-06     Completed Date:  1995-04-06     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  9007422     Medline TA:  Leuk Lymphoma     Country:  SWITZERLAND    
Other Details:
Languages:  eng     Pagination:  411-8     Citation Subset:  IM    
Affiliation:
Department of Pediatrics, Mie University School of Medicine, Japan.
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MeSH Terms
Descriptor/Qualifier:
Antigens, Surface / analysis*
Antineoplastic Agents / administration & dosage
Cell Division
Colony-Stimulating Factors / physiology
Cytokines / physiology*,  therapeutic use
Flow Cytometry
Hematopoietic Stem Cells / pathology
Humans
Immunophenotyping
Precursor Cell Lymphoblastic Leukemia-Lymphoma / pathology*,  therapy
Receptors, Cytokine / physiology*
Chemical
Reg. No./Substance:
0/Antigens, Surface; 0/Antineoplastic Agents; 0/Colony-Stimulating Factors; 0/Cytokines; 0/Receptors, Cytokine

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