Document Detail


Current strategies to minimize hepatic ischemia-reperfusion injury by targeting reactive oxygen species.
MedLine Citation:
PMID:  22459037     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Ischemia-reperfusion is a major component of injury in vascular occlusion both during liver surgery and during liver transplantation. The pathophysiology of hepatic ischemia-reperfusion includes a number of mechanisms including oxidant stress that contribute to various degrees to the overall organ damage. A large volume of recent research has focused on the use of antioxidants to ameliorate this injury, although results in experimental models have not translated well to the clinic. This review focuses on critical sources and mediators of oxidative stress during hepatic ischemia-reperfusion, the status of current antioxidant interventions, and emerging mechanisms of protection by preconditioning. While recent advances in regulation of antioxidant systems by Nrf2 provide interesting new potential therapeutic targets, an increased focus must be placed on more in-depth mechanistic investigations in hepatic ischemia-reperfusion injury and translational research in order to refine current strategies in disease management.
Authors:
Hartmut Jaeschke; Benjamin L Woolbright
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Review    
Journal Detail:
Title:  Transplantation reviews (Orlando, Fla.)     Volume:  26     ISSN:  1557-9816     ISO Abbreviation:  Transplant Rev (Orlando)     Publication Date:  2012 Apr 
Date Detail:
Created Date:  2012-03-30     Completed Date:  2012-08-06     Revised Date:  2013-04-03    
Medline Journal Info:
Nlm Unique ID:  8804364     Medline TA:  Transplant Rev (Orlando)     Country:  United States    
Other Details:
Languages:  eng     Pagination:  103-14     Citation Subset:  IM    
Copyright Information:
Copyright © 2012 Elsevier Inc. All rights reserved.
Affiliation:
Department of Pharmacology, Toxicology and Therapeutics, University of Kansas Medical Center, Kansas City, KS 66160, USA. hjaeschke@kumc.edu
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MeSH Terms
Descriptor/Qualifier:
Antioxidants / therapeutic use*
Apoptosis
Glutathione / therapeutic use
Heat-Shock Proteins / therapeutic use
Humans
Ischemic Preconditioning
Kupffer Cells / metabolism
Lipid Peroxidation
Liver / blood supply*
Mitochondria, Liver / metabolism
Neutrophils / metabolism
Oxidative Stress
Reactive Oxygen Species / metabolism*
Reperfusion Injury / metabolism,  physiopathology,  prevention & control*
Superoxide Dismutase / therapeutic use
Grant Support
ID/Acronym/Agency:
P20 RR016475/RR/NCRR NIH HHS; P20 RR021940/RR/NCRR NIH HHS; P20 RR021940-06/RR/NCRR NIH HHS; R01 AA012916-10/AA/NIAAA NIH HHS; R01 AA12916/AA/NIAAA NIH HHS; R01 DK070195/DK/NIDDK NIH HHS; R01 DK070195-05/DK/NIDDK NIH HHS; T32 ES007079-26A2/ES/NIEHS NIH HHS; T32 ES007079-32/ES/NIEHS NIH HHS
Chemical
Reg. No./Substance:
0/Antioxidants; 0/Heat-Shock Proteins; 0/Reactive Oxygen Species; 70-18-8/Glutathione; EC 1.15.1.1/Superoxide Dismutase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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