Document Detail


Current smokers develop more posterior myocardial infarctions probably due to increased tendency to thrombosis.
MedLine Citation:
PMID:  10816064     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
This investigation was carried out to determine whether smokers developed smaller infarcts as assessed by peak enzyme levels and also to what extent smoking could modify infarct localization. The study included 753 patients, of whom 351 had no history of previous coronary heart disease (CHD) (angina pectoris and/or myocardial infarction (MI)). The investigation was designed as an exposed (smoking) versus non-exposed (non-smoking) cohort study. Outcome was infarct size, posterior versus non-posterior MI and non-Q-wave versus Q-wave infarcts. In the total cohort of patients, 312 (41%) were smokers, the corresponding number in the restricted cohort of patients without a previous CHD (CHD-0-pts) was 169 (48%). Smokers were younger than non-smokers, and more of them were males. It was found that infarct size was similar in smokers and in non-smokers (crude and adjusted effects). Crude effects showed that smokers developed significantly more posterior infarcts than non-smokers; odds ratio (OR) for developing a posterior MI was 1.95 (2p < 0.001) (all patients) and 2.34 (2P < 0.001) (CHD-0-pts), respectively. After adjusting for confounders (logistic regression model), OR in the two groups was 1.24 (2p = 0.256) and 1.95 (2p = 0.01), respectively. The study shows that current smokers were younger, and indicates that in those without a previous CHD, significantly more of them developed a posterior MI.
Authors:
K Landmark; M Abdelnoor
Related Documents :
18805174 - Targeted myocardial microinjections of a biocomposite material reduces infarct expansio...
991264 - The clinical and angiographic spectrum of isolated, nondominant, left circumflex corona...
18195484 - Expression of sdf-1-cxcr4 axis and an anti-remodelling effectiveness of foetal-liver st...
17067624 - Acute anterior myocardial infarction followed by acute inferoposterior myocardial infar...
3366994 - Prognostic cardiac catheterization variables in survivors of acute myocardial infarctio...
18666844 - Tailored antithrombotic therapy for acute coronary syndromes.
Publication Detail:
Type:  Comparative Study; Journal Article    
Journal Detail:
Title:  Scandinavian cardiovascular journal : SCJ     Volume:  34     ISSN:  1401-7431     ISO Abbreviation:  Scand. Cardiovasc. J.     Publication Date:  2000  
Date Detail:
Created Date:  2000-07-14     Completed Date:  2000-07-14     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  9708377     Medline TA:  Scand Cardiovasc J     Country:  NORWAY    
Other Details:
Languages:  eng     Pagination:  73-8     Citation Subset:  IM    
Affiliation:
Department of Pharmacotherapeutics, Research Forum, Ullevål University Hospital, Oslo, Norway. k.h.landmark@ioks.uio.no
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Aged
Aged, 80 and over
Biological Markers / blood
Coronary Thrombosis / complications*,  enzymology,  epidemiology,  etiology
Creatine Kinase / blood
Electrocardiography
Female
Humans
Isoenzymes
L-Lactate Dehydrogenase / blood
Male
Middle Aged
Myocardial Infarction / enzymology,  epidemiology,  etiology*
Odds Ratio
Prevalence
Prognosis
Retrospective Studies
Smoking / adverse effects*,  blood,  epidemiology
Sweden / epidemiology
Chemical
Reg. No./Substance:
0/Biological Markers; 0/Isoenzymes; EC 1.1.1.27/L-Lactate Dehydrogenase; EC 2.7.3.2/Creatine Kinase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Limitation of physical activity, dyspnoea and chest pain before and two years after coronary artery ...
Next Document:  Atrial septal defect in adults. Thirty-eight-year follow-up of a surgically and a conservatively man...