Document Detail


Crosstalk between B lymphocytes, microbiota and the intestinal epithelium governs immunity versus metabolism in the gut.
MedLine Citation:
PMID:  22101768     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Using a systems biology approach, we discovered and dissected a three-way interaction between the immune system, the intestinal epithelium and the microbiota. We found that, in the absence of B cells, or of IgA, and in the presence of the microbiota, the intestinal epithelium launches its own protective mechanisms, upregulating interferon-inducible immune response pathways and simultaneously repressing Gata4-related metabolic functions. This shift in intestinal function leads to lipid malabsorption and decreased deposition of body fat. Network analysis revealed the presence of two interconnected epithelial-cell gene networks, one governing lipid metabolism and another regulating immunity, that were inversely expressed. Gene expression patterns in gut biopsies from individuals with common variable immunodeficiency or with HIV infection and intestinal malabsorption were very similar to those of the B cell-deficient mice, providing a possible explanation for a longstanding enigmatic association between immunodeficiency and defective lipid absorption in humans.
Authors:
Natalia Shulzhenko; Andrey Morgun; William Hsiao; Michele Battle; Michael Yao; Oksana Gavrilova; Marlene Orandle; Lloyd Mayer; Andrew J Macpherson; Kathy D McCoy; Claire Fraser-Liggett; Polly Matzinger
Related Documents :
21771898 - Dendritic cells in bacteria handling in the gut.
10088138 - Negative immunoregulatory effects of antidepressants: inhibition of interferon-gamma an...
7500028 - Regulation of the balance of cytokine production and the signal transducer and activato...
8384238 - Oxidative priming of neutrophils by interferon-gamma.
19415548 - Lipopolysaccharide stimulation of dendritic cells induces interleukin-10 producing alle...
21771898 - Dendritic cells in bacteria handling in the gut.
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural     Date:  2011-11-20
Journal Detail:
Title:  Nature medicine     Volume:  17     ISSN:  1546-170X     ISO Abbreviation:  Nat. Med.     Publication Date:  2011 Dec 
Date Detail:
Created Date:  2011-12-07     Completed Date:  2012-02-02     Revised Date:  2012-05-03    
Medline Journal Info:
Nlm Unique ID:  9502015     Medline TA:  Nat Med     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1585-93     Citation Subset:  IM    
Affiliation:
'Ghost Lab', T Cell Tolerance and Memory Section, Laboratory of Cellular and Molecular Immunology, National Institute of Allergy and Infectious Diseases (NIAID), US National Institutes of Health (NIH), Bethesda, Maryland, USA. natalia.shulzhenko@oregonstate.edu
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
B-Lymphocytes / immunology*,  metabolism
DNA, Bacterial / isolation & purification
Epithelial Cells / immunology,  microbiology
GATA4 Transcription Factor / genetics,  metabolism
Gene Expression Profiling
Gene Regulatory Networks
Humans
Immunoglobulin A / immunology
Intestinal Mucosa / immunology*,  metabolism,  microbiology*
Lipid Metabolism*
Metagenome*
Mice
Mice, Inbred BALB C
Mice, Knockout
Microarray Analysis
Up-Regulation
Grant Support
ID/Acronym/Agency:
P40RR018603/RR/NCRR NIH HHS; R01 DK087873/DK/NIDDK NIH HHS; U24 DK059630/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/DNA, Bacterial; 0/GATA4 Transcription Factor; 0/Gata4 protein, mouse; 0/Immunoglobulin A
Comments/Corrections
Comment In:
Nat Med. 2011 Dec;17(12):1549-50   [PMID:  22146454 ]
Nat Rev Immunol. 2012 Jan;12(1):2   [PMID:  22193762 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Mesenchymal stem cell-based tissue regeneration is governed by recipient T lymphocytes via IFN-? and...
Next Document:  A mechanism for glycoconjugate vaccine activation of the adaptive immune system and its implications...