| Creation of a one-way interatrial communication in the treatment of critical pulmonary stenosis with intact ventricular septum: a case report. | |
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MedLine Citation:
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PMID: 8969384 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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BACKGROUND: In biventricular repair of pulmonary outflow tract obstruction with intact ventricular septum, the right ventricle is loaded with total pulmonary blood flow acutely as the right-to-left shunt is abolished by closure of the atrial septal defect (ASD). METHODS: We designed a one-way interatrial communication by creation of an atrial septal flap to reduce the excessive volume load of the right ventricle. RESULTS: This procedure was successfully performed in a 3-year-old girl undergoing definitive biventricular repair for critical pulmonary stenosis associated with tricuspid stenosis and a small right ventricle. CONCLUSIONS: We believe that creation of a one-way interatrial communication might be a good alternative to adjustable ASD and/or bidirectional Glenn shunt in biventricular repair of critical pulmonary stenosis or pulmonary atresia with intact ventricular septum. |
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Authors:
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S F Lin; I S Chiu; R B Hsu |
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Publication Detail:
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Type: Case Reports; Journal Article |
Journal Detail:
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Title: Journal of cardiac surgery Volume: 11 ISSN: 0886-0440 ISO Abbreviation: J Card Surg Publication Date: 1996 Sep-Oct |
Date Detail:
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Created Date: 1997-02-27 Completed Date: 1997-02-27 Revised Date: 2004-11-17 |
Medline Journal Info:
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Nlm Unique ID: 8908809 Medline TA: J Card Surg Country: UNITED STATES |
Other Details:
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Languages: eng Pagination: 368-70 Citation Subset: IM |
Affiliation:
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Department of Surgery, National Taiwan University Hospital, Taipei. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Child, Preschool Echocardiography Female Heart Septal Defects, Atrial / complications, surgery Humans Pulmonary Valve Stenosis / etiology, surgery*, ultrasonography |
| Comments/Corrections | |
Comment In:
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J Card Surg. 1997 May-Jun;12(3):202-3
[PMID:
9395952
]
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From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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