Document Detail


Coupling between hydrodynamic forces and planar cell polarity orients mammalian motile cilia.
MedLine Citation:
PMID:  20305650     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
In mammals, motile cilia cover many organs, such as fallopian tubes, respiratory tracts and brain ventricles. The development and function of these organs critically depend on efficient directional fluid flow ensured by the alignment of ciliary beating. To identify the mechanisms involved in this process, we analysed motile cilia of mouse brain ventricles, using biophysical and molecular approaches. Our results highlight an original orientation mechanism for ependymal cilia whereby basal bodies first dock apically with random orientations, and then reorient in a common direction through a coupling between hydrodynamic forces and the planar cell polarity (PCP) protein Vangl2, within a limited time-frame. This identifies a direct link between external hydrodynamic cues and intracellular PCP signalling. Our findings extend known PCP mechanisms by integrating hydrodynamic forces as long-range polarity signals, argue for a possible sensory role of ependymal cilia, and will be of interest for the study of fluid flow-mediated morphogenesis.
Authors:
Boris Guirao; Alice Meunier; St?phane Mortaud; Andrea Aguilar; Jean-Marc Corsi; Laetitia Strehl; Yuki Hirota; Ang?lique Desoeuvre; Camille Boutin; Young-Goo Han; Zaman Mirzadeh; Harold Cremer; Mireille Montcouquiol; Kazunobu Sawamoto; Nathalie Spassky
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-03-21
Journal Detail:
Title:  Nature cell biology     Volume:  12     ISSN:  1476-4679     ISO Abbreviation:  Nat. Cell Biol.     Publication Date:  2010 Apr 
Date Detail:
Created Date:  2010-04-02     Completed Date:  2010-04-26     Revised Date:  2010-05-05    
Medline Journal Info:
Nlm Unique ID:  100890575     Medline TA:  Nat Cell Biol     Country:  England    
Other Details:
Languages:  eng     Pagination:  341-50     Citation Subset:  IM    
Affiliation:
Institut de Biologie de l'Ecole Normale Sup?rieure (IBENS), Institut National de la Sant? et de la Recherche M?dicale U1024, Centre National de la Recherche Scientifique UMR8197, 75005 Paris, France.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Polarity*
Cells, Cultured
Cerebrospinal Fluid / metabolism
Cilia / metabolism
Ependyma / cytology*,  embryology,  metabolism
Feedback, Physiological
Humans
Kinesin / metabolism
Mechanotransduction, Cellular*
Mice
Mice, Transgenic
Morphogenesis
Motion
Mutation
Nerve Tissue Proteins / genetics,  metabolism*
Recombinant Fusion Proteins / metabolism
Stress, Mechanical
Time Factors
Transfection
Tumor Suppressor Proteins / metabolism
Chemical
Reg. No./Substance:
0/Kif3a protein, mouse; 0/Ltap protein, mouse; 0/Nerve Tissue Proteins; 0/Recombinant Fusion Proteins; 0/Tg737Rpw protein, mouse; 0/Tumor Suppressor Proteins; EC 3.6.1.-/Kinesin
Comments/Corrections
Comment In:
Nat Cell Biol. 2010 Apr;12(4):314-5   [PMID:  20360747 ]
Nat Rev Mol Cell Biol. 2010 May;11(5):313   [PMID:  20419890 ]
Erratum In:
Nat Cell Biol. 2010 May;12(5):520

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