Document Detail


Could sudden cardiac death in epilepsy be related to the occurrence of thalamic dysfunction or anatomic change?
MedLine Citation:
PMID:  19330235     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Sudden unexpected death in epilepsy (SUDEP) is the most important direct epilepsy-related cause of death in people with chronic epilepsy. Its physiopathology is still unknown; however, the most commonly suggested potential mechanisms involve cardiac or respiratory abnormalities. As the anatomical substrate of epileptic activity in the central nervous system (CNS) shows a direct relationship with cardiovascular alterations, this may suggests that patients with epilepsy associated with focal CNS lesions may be at particular risk of SUDEP. Currently, experimental and clinical data support an important role for thalamic nuclei in the behavioural manifestations, initiation and propagation of seizures. In view of the above findings, we purpose that SUDEP, at least in some cases, could be related to the occurrence of thalamic dysfunction or anatomic change.
Authors:
Fulvio A Scorza; Lineu Calderazzo; Marly de Albuquerque; Ricardo M Arida; Antonio-Carlos G de Almeida; Esper A Cavalheiro
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Arquivos de neuro-psiquiatria     Volume:  67     ISSN:  1678-4227     ISO Abbreviation:  Arq Neuropsiquiatr     Publication Date:  2009 Mar 
Date Detail:
Created Date:  2009-03-30     Completed Date:  2010-02-11     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0125444     Medline TA:  Arq Neuropsiquiatr     Country:  Brazil    
Other Details:
Languages:  eng     Pagination:  139-43     Citation Subset:  IM    
Affiliation:
Disciplina de Neurologia Experimental, Escola Paulista de Medicina, Universidade Federal de S?o Paulo, S?o Paulo, SP, Brasil. scorza.nexp@epm.br
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MeSH Terms
Descriptor/Qualifier:
Death, Sudden, Cardiac* / etiology,  pathology
Epilepsy* / complications,  physiopathology
Heart Rate / physiology
Humans
Risk Factors
Thalamic Diseases* / complications,  physiopathology
Thalamus* / pathology,  physiopathology

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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