Document Detail

Coronary slow flow and acute coronary syndrome in a patient with spinal cord injury.
MedLine Citation:
PMID:  21841878     Owner:  NLM     Status:  MEDLINE    
We report the case of a 55-year-old man who presented with acute coronary syndrome due to coronary slow flow after spinal cord injury. Data regarding the causes and clinical manifestations of coronary slow flow are inconclusive, but the autonomic nervous system is believed to be at least a contributing factor. The predominant vagal activity causes vasodilation and hemostasis, which can lead to acute coronary syndrome. We hereby call attention to hyperactive parasympathetic tonicity, which can lead to coronary slow flow and acute coronary syndrome in acute spinal cord injury patients.
Meryem Aktoz; Ersan Tatli; Ahmet Barutcu; Flora Ozkalayci; Elif Umit; Armagan Altun
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Publication Detail:
Type:  Case Reports; Journal Article; Video-Audio Media    
Journal Detail:
Title:  Texas Heart Institute journal / from the Texas Heart Institute of St. Luke's Episcopal Hospital, Texas Children's Hospital     Volume:  38     ISSN:  1526-6702     ISO Abbreviation:  Tex Heart Inst J     Publication Date:  2011  
Date Detail:
Created Date:  2011-08-15     Completed Date:  2012-01-05     Revised Date:  2013-06-28    
Medline Journal Info:
Nlm Unique ID:  8214622     Medline TA:  Tex Heart Inst J     Country:  United States    
Other Details:
Languages:  eng     Pagination:  433-6     Citation Subset:  IM    
Department of Cardiology, Trakya University, 22030 Edirne, Turkey.
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MeSH Terms
Acute Coronary Syndrome / diagnosis,  drug therapy,  etiology*,  physiopathology
Cardiovascular Agents / therapeutic use
Coronary Angiography
Coronary Circulation* / drug effects
Magnetic Resonance Imaging
Middle Aged
No-Reflow Phenomenon / diagnosis,  drug therapy,  etiology*,  physiopathology
Physical Therapy Modalities
Spinal Cord Injuries / complications*,  diagnosis,  physiopathology,  therapy
Treatment Outcome
Vagus Nerve / physiopathology
Reg. No./Substance:
0/Cardiovascular Agents

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine

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