Document Detail


Coronary flow as a determinant of c-myc and c-fos proto-oncogene expression in an isolated adult rat heart.
MedLine Citation:
PMID:  3135413     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Chronic cardiac overload induces quantitative and qualitative changes of the phenotype which finally adapt the myocardium to its new functional requirements (Swynghedauw, 1986). It has been proposed that both stretch and enhanced isometric tension could trigger these modifications (Peterson and Lesch, 1972), but until now no real messenger has been found. In a search for signals which may account for these changes, we decided to investigate the expression of two proto-oncogenes, c-fos and c-myc, coding for nuclear proteins (review in Adamson, 1987) because several of their properties are consistent with their possessing a role in the transduction of extracellular growth signals to the cell interior. We report here that, in adult rat heart, expression of the c-fos and c-myc proto-oncogene was both sequentially and transitorily increased when, in a beating heart but not in an arrested heart, the coronary flow (and/or pressure) was augmented. This was studied in an isolated, coronary perfused heart preparation, a model in which the initial conditions of a cardiac overload may be mimicked in such a way that protein synthesis is stimulated by increasing the coronary perfusion pressure (Kira et al., 1984).
Authors:
C Bauters; J M Moalic; J Bercovici; C Mouas; R Emanoil-Ravier; S Schiaffino; B Swynghedauw
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Journal of molecular and cellular cardiology     Volume:  20     ISSN:  0022-2828     ISO Abbreviation:  J. Mol. Cell. Cardiol.     Publication Date:  1988 Feb 
Date Detail:
Created Date:  1988-08-30     Completed Date:  1988-08-30     Revised Date:  2003-11-14    
Medline Journal Info:
Nlm Unique ID:  0262322     Medline TA:  J Mol Cell Cardiol     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  97-101     Citation Subset:  IM    
Affiliation:
U 127 INSERM, Hopital Lariboisière, Paris, France.
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MeSH Terms
Descriptor/Qualifier:
Animals
Coronary Circulation*
Myocardial Contraction*
Myocardium / metabolism*
Perfusion
Proto-Oncogene Proteins / biosynthesis*
Proto-Oncogene Proteins c-fos
Proto-Oncogene Proteins c-myc
Rats
Chemical
Reg. No./Substance:
0/Proto-Oncogene Proteins; 0/Proto-Oncogene Proteins c-fos; 0/Proto-Oncogene Proteins c-myc

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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