Document Detail


Cordyceps pruinosa extracts induce apoptosis of HeLa cells by a caspase dependent pathway.
MedLine Citation:
PMID:  20138133     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
AIM OF THE STUDY: Cordyceps is a parasitic fungus and has long been used as a traditional Chinese medicine to treat illnesses, promote longevity, increase athletic power, and relieve exhaustion and cancer. In this study, we reveal the mechanisms underlying apoptosis induced by Cordyceps pruinosa butanol fraction (CPBF) in the human cervical adenocarcinoma cell line, HeLa. MATERIALS AND METHODS: Proliferation and apoptosis of cells were examined by MTT assay, DNA fragmentation, phosphatidyl serine distribution assay, Western blot analysis, and immunocytochemistry. To determine the association between CPBF related apoptosis and ROS, electron spin resonance (ESR) trapping experiments were used. RESULTS: CPBF inhibited proliferation and induced apoptosis in HeLa cells in a dose-dependent manner using a MTT assay, DNA fragmentation, and a phosphatidyl serine distribution assay. Western blot analysis showed that apoptosis in HeLa cells was caspase-3- and -9-dependent. Proteolytic cleavage of PARP and the release of cytochrome c from the mitochondria into the cytosol were significantly increased and the Bcl-2/Bax protein ratio was decreased. Apoptosis induced by CPBF was not prevented by various antioxidants. CONCLUSIONS: These results indicate that apoptotic effects of CPBF on HeLa cells are mediated by mitochondria-dependent death-signaling pathway independent of reactive oxygen species, suggesting that CPBF might be effective as an anti-proliferative agent for cancer.
Authors:
Ho Gyoung Kim; Heesang Song; Deok Hyo Yoon; Byeong-Wook Song; Sang Min Park; Gi Ho Sung; Jae-Youl Cho; Hae Il Park; Sunga Choi; Won O Song; Ki-Chul Hwang; Tae Woong Kim
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-02-04
Journal Detail:
Title:  Journal of ethnopharmacology     Volume:  128     ISSN:  1872-7573     ISO Abbreviation:  J Ethnopharmacol     Publication Date:  2010 Mar 
Date Detail:
Created Date:  2010-03-09     Completed Date:  2010-07-01     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  7903310     Medline TA:  J Ethnopharmacol     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  342-51     Citation Subset:  IM    
Copyright Information:
Copyright (c) 2010 Elsevier Ireland Ltd. All rights reserved.
Affiliation:
Department of Biochemistry, Kangwon National University, Chuncheon 200-701, Republic of Korea.
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MeSH Terms
Descriptor/Qualifier:
Apoptosis / drug effects*
Caspase 3 / metabolism*
Caspases / metabolism*
Cordyceps / metabolism*
Cytochrome c Group / metabolism
Cytochromes c / metabolism
DNA Fragmentation / drug effects
Hela Cells
Humans
Mitochondria / metabolism
Plant Extracts / pharmacology*
Poly(ADP-ribose) Polymerases / metabolism
Reactive Oxygen Species / metabolism
Signal Transduction / drug effects
bcl-2-Associated X Protein / metabolism
Chemical
Reg. No./Substance:
0/Cytochrome c Group; 0/Plant Extracts; 0/Reactive Oxygen Species; 0/bcl-2-Associated X Protein; 116110-46-4/cytochrome c''; 9007-43-6/Cytochromes c; EC 2.4.2.30/Poly(ADP-ribose) Polymerases; EC 3.4.22.-/Caspase 3; EC 3.4.22.-/Caspases

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