Document Detail


Cooperation between heparin-binding EGF-like growth factor and interleukin-6 in promoting the growth of human myeloma cells.
MedLine Citation:
PMID:  11971193     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Interleukin-6 (IL-6) is a major survival and proliferation factor of human malignant plasma cells and IL-6 dependent myeloma cell lines can be obtained from patients with terminal disease. We show here that mutated diphtheria toxin, a specific inhibitor of heparin-binding epidermal growth factor-like growth factor (HB-EGF), blocked the IL-6-induced growth of two myeloma cell lines (XG-1 and XG-14) and did not significantly affect that of two other cell lines (XG-6 and XG-13). The IL-6 mediated growth of myeloma cells was also inhibited by antibodies to ErbB1, a receptor for HB-EGF. The XG-1 and XG-14 cell lines that are sensitive to HB-EGF inhibitors overexpressed HB-EGF and EGF receptor (ErbB1) genes. They also overexpressed CD9, a tetraspanin that binds to the heparin-binding domain of HB-EGF and is critical for promoting ErbB1 activation by HB-EGF. The XG-6 and XG-13 myeloma cells that were not significantly sensitive to HB-EGF antagonists, poorly expressed HB-EGF, ErbB1 and CD9 genes or proteins. We demonstrated that recombinant HB-EGF supported the long-term growth of myeloma cells, as did IL-6. The myeloma cell growth factor activity of HB-EGF was completely inhited by antibodies to ErbB1, but also by antibodies to gp130 IL-6 transducer or to IL-6. These data indicate that in the XG-1 and XG-14 IL-6-dependent myeloma cell lines, the CD9/HB-EGF/erbB1 and the IL-6/IL-6R/gp130 pathways cooperate synergistically to trigger myeloma cell growth. They suggest that inhibitors of the EGF receptor or HB-EGF may be useful for inducing myeloma cell apoptosis in patients with multiple myeloma.
Authors:
Yue Dan Wang; John De Vos; Michel Jourdan; Guilhem Couderc; Zhao-Yang Lu; Jean-François Rossi; Bernard Klein
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Oncogene     Volume:  21     ISSN:  0950-9232     ISO Abbreviation:  Oncogene     Publication Date:  2002 Apr 
Date Detail:
Created Date:  2002-04-23     Completed Date:  2002-05-08     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  8711562     Medline TA:  Oncogene     Country:  England    
Other Details:
Languages:  eng     Pagination:  2584-92     Citation Subset:  IM    
Affiliation:
INSERM U475 and Unit for Cellular Therapy, CHU Montpellier, 99 Rue Puech Villa, 34197 Montpellier, France.
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MeSH Terms
Descriptor/Qualifier:
Antigens, CD / metabolism
Apoptosis
Autocrine Communication
Cell Division / drug effects
Cell Survival / drug effects
Dose-Response Relationship, Drug
Drug Synergism
Epidermal Growth Factor / pharmacology*
Humans
Intercellular Signaling Peptides and Proteins
Interleukin-6 / pharmacology*
Kinetics
Membrane Glycoproteins*
Models, Biological
Multiple Myeloma / metabolism,  pathology*
RNA, Neoplasm / biosynthesis
Receptor, Epidermal Growth Factor / genetics,  metabolism
Tumor Cells, Cultured
Chemical
Reg. No./Substance:
0/Antigens, CD; 0/CD9 antigen; 0/Intercellular Signaling Peptides and Proteins; 0/Interleukin-6; 0/Membrane Glycoproteins; 0/RNA, Neoplasm; 149176-25-0/heparin-binding EGF-like growth factor; 62229-50-9/Epidermal Growth Factor; EC 2.7.10.1/Receptor, Epidermal Growth Factor

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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