Document Detail


Control of glucose homeostasis and insulin sensitivity by the Let-7 family of microRNAs.
MedLine Citation:
PMID:  22160727     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Diabetes mellitus is the most common metabolic disorder worldwide and a major risk factor for cardiovascular disease. MicroRNAs are negative regulators of gene expression that have been implicated in many biological processes, including metabolism. Here we show that the Let-7 family of microRNAs regulates glucose metabolism in multiple organs. Global and pancreas-specific overexpression of Let-7 in mice resulted in impaired glucose tolerance and reduced glucose-induced pancreatic insulin secretion. Mice overexpressing Let-7 also had decreased fat mass and body weight, as well as reduced body size. Global knockdown of the Let-7 family with an antimiR was sufficient to prevent and treat impaired glucose tolerance in mice with diet-induced obesity, at least in part by improving insulin sensitivity in liver and muscle. AntimiR treatment of mice on a high-fat diet also resulted in increased lean and muscle mass, but not increased fat mass, and prevented ectopic fat deposition in the liver. These findings demonstrate that Let-7 regulates multiple aspects of glucose metabolism and suggest antimiR-induced Let-7 knockdown as a potential treatment for type 2 diabetes mellitus. Furthermore, our Cre-inducible Let-7-transgenic mice provide a unique model for studying tissue-specific aspects of body growth and type 2 diabetes.
Authors:
Robert J A Frost; Eric N Olson
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2011-12-12
Journal Detail:
Title:  Proceedings of the National Academy of Sciences of the United States of America     Volume:  108     ISSN:  1091-6490     ISO Abbreviation:  Proc. Natl. Acad. Sci. U.S.A.     Publication Date:  2011 Dec 
Date Detail:
Created Date:  2011-12-28     Completed Date:  2012-02-21     Revised Date:  2012-03-29    
Medline Journal Info:
Nlm Unique ID:  7505876     Medline TA:  Proc Natl Acad Sci U S A     Country:  United States    
Other Details:
Languages:  eng     Pagination:  21075-80     Citation Subset:  IM    
Affiliation:
Department of Molecular Biology, University of Texas Southwestern Medical Center, Dallas, TX 75390, USA. robert.frost@utsouthwestern.edu
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MeSH Terms
Descriptor/Qualifier:
Analysis of Variance
Animals
Blotting, Northern
Blotting, Western
Body Composition
Body Weight
Diabetes Mellitus, Type 2 / metabolism*
Gene Expression Regulation / physiology
Gene Knockdown Techniques
Glucose / metabolism*
Homeostasis / physiology*
Insulin Resistance / physiology*
Liver / metabolism
Mice
Mice, Inbred C57BL
Mice, Obese
Mice, Transgenic
MicroRNAs / genetics,  metabolism*
Grant Support
ID/Acronym/Agency:
R01 HL093039-04/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/MicroRNAs; 0/mirnlet7 microRNA, mouse; 50-99-7/Glucose

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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