Document Detail

Consumption of green tea favorably affects oxidative stress markers in weight-trained men.
MedLine Citation:
PMID:  18337059     Owner:  NLM     Status:  MEDLINE    
OBJECTIVE: This study investigated the effects of the consumption of green tea (GT) for 7 d on biomarkers of oxidative stress in young men undergoing resistance exercise. METHODS: Fourteen subjects performed a bench press exercise (four sets, 10 to 4 repetitions) after undergoing a period without (control group) or with the intake of GT (GT group; 2 g of leaves in 200 mL of water, three times per day). Blood samples were obtained before and after exercise and analyzed for total antioxidant capacity (ferric reducing ability of plasma [FRAP]), total polyphenols, reduced glutathione (GSH), lipid hydroperoxide (LH) and thiobarbituric acid-reactive substances, creatine kinase (CK), aspartate aminotransferase (AST), xanthine oxidase (XO), hypoxanthine, and uric acid (UA). RESULTS: In the control group, exercise did not affect the values of LH, thiobarbituric acid-reactive substances, and FRAP, although it did reduce the levels of GSH (P < 0.05). In addition, exercise increased CK, AST, and XO activities, although it did not change the values for hypoxanthine or UA. Green tea reduced the postexercise concentration of LH and increased the values of total polyphenols, GSH, and FRAP. GT also inhibited a significant rise in CK and XO activities induced by exercise. Furthermore, GT decreased the AST activity and hypoxanthine and UA concentrations before and after exercise. The assessment of food consumption revealed that the participants had an unbalanced diet, particularly in relation to vitamin E and carotenoids. CONCLUSION: Consumption of GT, a beverage rich in polyphenols, may offer protection against the oxidative damage caused by exercise, and dietary guidance for sports participants should be emphasized.
Vilma Simões Pereira Panza; Elisabeth Wazlawik; Gustavo Ricardo Schütz; Leandro Comin; Karl Christian Hecht; Edson Luiz da Silva
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Publication Detail:
Type:  Journal Article; Randomized Controlled Trial; Research Support, Non-U.S. Gov't     Date:  2008-03-12
Journal Detail:
Title:  Nutrition (Burbank, Los Angeles County, Calif.)     Volume:  24     ISSN:  0899-9007     ISO Abbreviation:  Nutrition     Publication Date:  2008 May 
Date Detail:
Created Date:  2008-04-11     Completed Date:  2008-08-11     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8802712     Medline TA:  Nutrition     Country:  United States    
Other Details:
Languages:  eng     Pagination:  433-42     Citation Subset:  IM    
Department of Nutrition, Federal University of Santa Catarina, Florianópolis, Santa Catarina, Brazil.
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MeSH Terms
Antioxidants / analysis,  pharmacology*
Aspartate Aminotransferases / metabolism
Biological Markers / analysis,  metabolism
Creatine Kinase / metabolism
Cross-Over Studies
Exercise / physiology
Flavonoids / analysis,  pharmacology*
Glutathione / metabolism
Hypoxanthine / metabolism
L-Lactate Dehydrogenase / metabolism
Lipid Peroxidation / drug effects*
Oxidative Stress
Oxygen Consumption
Phenols / analysis,  pharmacology*
Tea* / chemistry
Thiobarbituric Acid Reactive Substances / metabolism
Uric Acid / metabolism
Weight Lifting / physiology*
Xanthine Oxidase / metabolism
Reg. No./Substance:
0/Antioxidants; 0/Biological Markers; 0/Flavonoids; 0/Phenols; 0/Tea; 0/Thiobarbituric Acid Reactive Substances; 0/polyphenols; 68-94-0/Hypoxanthine; 69-93-2/Uric Acid; 70-18-8/Glutathione; EC Dehydrogenase; EC Oxidase; EC Aminotransferases; EC Kinase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine

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