Document Detail


Connexin 26 (GJB2) mutations as a cause of the KID syndrome with hearing loss.
MedLine Citation:
PMID:  20307501     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
KID syndrome (MIM 148210) is an ectodermal dysplasia characterized by the occurrence of localized erythematous scaly skin lesions, keratitis and severe bilateral sensorineural deafness. KID syndrome is inherited as an autosomic dominant disease, due to mutations in the gene encoding gap junction protein GJB2 (connexin 26, Cx26). Cx26 is a component of gap junction channels in the epidermis and in the stria vascularis of the cochlea. These channels play a role in the coordinated exchange of molecules and ions occurring in a wide spectrum of cellular activities. In this paper we describe two patients with Cx26 mutations cause cell death by the alteration of protein trafficking, membrane localization and probably interfering with intracellular ion concentrations. We discuss the pathogenesis of both the hearing and skin phenotypes.
Authors:
Alessandro Terrinoni; Andrea Codispoti; Valeria Serra; Ernesto Bruno; Biagio Didona; Mauro Paradisi; Steven Nisticò; Elena Campione; Bianca Napolitano; Laura Diluvio; Gerry Melino
Publication Detail:
Type:  Case Reports; Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-03-20
Journal Detail:
Title:  Biochemical and biophysical research communications     Volume:  395     ISSN:  1090-2104     ISO Abbreviation:  Biochem. Biophys. Res. Commun.     Publication Date:  2010 Apr 
Date Detail:
Created Date:  2010-04-26     Completed Date:  2010-05-07     Revised Date:  2011-11-24    
Medline Journal Info:
Nlm Unique ID:  0372516     Medline TA:  Biochem Biophys Res Commun     Country:  United States    
Other Details:
Languages:  eng     Pagination:  25-30     Citation Subset:  IM    
Copyright Information:
Crown Copyright 2010. Published by Elsevier Inc. All rights reserved.
Affiliation:
Biochemistry Laboratory, IDI-IRCCS, C/O Department of Experimental Medicine and Biochemical Sciences, University of Rome Tor Vergata, 00133 Rome, Italy. alessandro.terrinoni@uniroma2.it
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MeSH Terms
Descriptor/Qualifier:
Apoptosis / genetics
Child
Connexins / genetics*,  metabolism
DNA Mutational Analysis
Ectodermal Dysplasia / genetics*,  metabolism,  pathology
Female
Hearing Loss / genetics*,  metabolism
Humans
Ichthyosis / genetics*,  metabolism,  pathology
Male
Protein Transport
Syndrome
Young Adult
Grant Support
ID/Acronym/Agency:
GGP09133//Telethon; //Medical Research Council
Chemical
Reg. No./Substance:
0/Connexins; 127120-53-0/connexin 26

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