Document Detail


Connecting the early brain injury of aneurysmal subarachnoid hemorrhage to clinical practice.
MedLine Citation:
PMID:  20401843     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Aneurysmal subarachnoid hemorrhage (SAH) is a devastating neurological disease that has a mortality rate as high as 67% in some series. Traditional research and treatment has focused on addressing the delayed events of cerebral vasospasm following SAH. However, the physiological and cellular events of early brain injury (EBI) make significant contributions to patient outcomes and may even be a more significant factor than delayed cerebral vasospasm. EBI is the result of physiological derangements such as increased intracranial pressure (ICP), decreased cerebral blood flow (CBF), and global cerebral ischemia, which results in blood brain barrier dysfunction, inflammation, and oxidative cascades that lead to neuronal cell death. The consequence of these events to the patient is often death or significant neurological disability. The link between EBI and outcome has come under intense focus with recent studies failing to show improved outcomes following significant inhibition of cerebral vasospasm, and research into the inhibition of EBI cascades is being perused as an effective means of treating SAH patients.
Authors:
Robert Ayer; John Zhang
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Turkish neurosurgery     Volume:  20     ISSN:  1019-5149     ISO Abbreviation:  Turk Neurosurg     Publication Date:  2010 Apr 
Date Detail:
Created Date:  2010-04-19     Completed Date:  2010-09-30     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9423821     Medline TA:  Turk Neurosurg     Country:  Turkey    
Other Details:
Languages:  eng     Pagination:  159-66     Citation Subset:  IM    
Affiliation:
Loma Linda University Medical Center, Neurosurgery, Loma Linda, CA, USA.
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MeSH Terms
Descriptor/Qualifier:
Apoptosis
Brain Edema / mortality,  pathology
Brain Injuries / mortality*,  pathology*
Cerebral Infarction / mortality,  pathology
Cerebrovascular Circulation
Encephalitis / mortality,  pathology
Humans
Intracranial Hypertension / mortality,  pathology
Oxidative Stress
Subarachnoid Hemorrhage / mortality*,  pathology*
Vasospasm, Intracranial / mortality,  pathology

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