Document Detail


Compensatory phospholipid digestion is required for cholesterol absorption in pancreatic phospholipase A(2)-deficient mice.
MedLine Citation:
PMID:  11266383     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND AND AIMS: Numerous studies have suggested phospholipid inhibition of dietary cholesterol absorption through the gastrointestinal tract. This study addressed the importance of luminal phospholipid hydrolysis in this process. METHODS: The effect of phospholipase inhibition on cholesterol transport from intestinal lumen to the lymphatics was evaluated in lymph fistula rats. Cholesterol and phospholipid absorption efficiency in intact animals was evaluated in control and phospholipase A(2) (PLA2) gene-targeted mice. RESULTS: The PLA2 inhibitor FPL 67047XX retarded cholesterol absorption in a lymph fistula rat model. Under basal chow-fed dietary conditions, cholesterol absorption efficiency from a single bolus meal, and plasma lipid levels, were similar among PLA2+/+, PLA2+/-, and PLA2-/- mice. Interestingly, the nonhydrolyzable phospholipid dioleoyl ether phosphatidylcholine suppressed cholesterol absorption by 10% to 18% in mice without regard to their PLA2 genotype. When 1-palmitoyl-2-[(14)C]oleoyl-phosphatidylcholine was used as the substrate, the radiolabeled phospholipid was found to be hydrolyzed and absorbed with equal efficiency in PLA2+/+, PLA2+/-, and PLA2-/- mice. CONCLUSIONS: These results suggested that although phospholipid digestion in the intestinal lumen is a prerequisite for efficient cholesterol absorption, additional enzyme(s) can compensate for pancreatic PLA2 in catalyzing phospholipid digestion and facilitating cholesterol absorption in PLA2 knockout mice.
Authors:
B L Richmond; A C Boileau; S Zheng; K W Huggins; N A Granholm; P Tso; D Y Hui
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Gastroenterology     Volume:  120     ISSN:  0016-5085     ISO Abbreviation:  Gastroenterology     Publication Date:  2001 Apr 
Date Detail:
Created Date:  2001-03-27     Completed Date:  2001-04-12     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  0374630     Medline TA:  Gastroenterology     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1193-202     Citation Subset:  AIM; IM    
Affiliation:
Department of Pathology and Laboratory Medicine, University of Cincinnati College of Medicine, 231 Albert Sabin Way, Cincinnati, Ohio 45267-0529, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Carbon Radioisotopes / diagnostic use
Cholesterol, Dietary / pharmacokinetics*
Digestive System Fistula / metabolism
Female
Intestinal Absorption / physiology*
Lymph / metabolism
Male
Mice
Mice, Inbred C3H
Mice, Inbred C57BL
Mice, Knockout
Phospholipases A / genetics*,  metabolism
Phospholipases A2
Pregnancy
Rats
Rats, Sprague-Dawley
Triglycerides / pharmacokinetics
Grant Support
ID/Acronym/Agency:
DK54504/DK/NIDDK NIH HHS; F32 DK10065/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Carbon Radioisotopes; 0/Cholesterol, Dietary; 0/Triglycerides; EC 3.1.1.-/Phospholipases A; EC 3.1.1.4/Phospholipases A2

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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