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Common variation in GPC5 is associated with acquired nephrotic syndrome.
MedLine Citation:
PMID:  21441931     Owner:  NLM     Status:  Publisher    
Abstract/OtherAbstract:
Severe proteinuria is a defining factor of nephrotic syndrome irrespective of the etiology. Investigation of congenital nephrotic syndrome has shown that dysfunction of glomerular epithelial cells (podocytes) plays a crucial role in this disease. Acquired nephrotic syndrome is also assumed to be associated with podocyte injury. Here we identify an association between variants in GPC5, encoding glypican-5, and acquired nephrotic syndrome through a genome-wide association study and replication analysis (P value under a recessive model (P(rec)) = 6.0 × 10(-11), odds ratio = 2.54). We show that GPC5 is expressed in podocytes and that the risk genotype is associated with higher expression. We further show that podocyte-specific knockdown and systemic short interfering RNA injection confers resistance to podocyte injury in mouse models of nephrosis. This study identifies GPC5 as a new susceptibility gene for nephrotic syndrome and implicates GPC5 as a promising therapeutic target for reducing podocyte vulnerability in glomerular disease.
Authors:
Koji Okamoto; Katsushi Tokunaga; Kent Doi; Toshiro Fujita; Hodaka Suzuki; Tetsuo Katoh; Tsuyoshi Watanabe; Nao Nishida; Akihiko Mabuchi; Atsushi Takahashi; Michiaki Kubo; Shiro Maeda; Yusuke Nakamura; Eisei Noiri
Publication Detail:
Type:  JOURNAL ARTICLE     Date:  2011-3-27
Journal Detail:
Title:  Nature genetics     Volume:  -     ISSN:  1546-1718     ISO Abbreviation:  -     Publication Date:  2011 Mar 
Date Detail:
Created Date:  2011-3-28     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9216904     Medline TA:  Nat Genet     Country:  -    
Other Details:
Languages:  ENG     Pagination:  -     Citation Subset:  -    
Affiliation:
1] Department of Nephrology and Endocrinology, Department of Hemodialysis and Apheresis, University Hospital, The University of Tokyo, Tokyo, Japan. [2] Department of Human Genetics, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan.
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