Document Detail


Common mechanisms in immune-mediated inflammatory disease.
MedLine Citation:
PMID:  15693110     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Characterization of the K/BxN mouse model of spontaneous arthritis contributed to the rediscovery of immune complex-mediated inflammation in rheumatoid arthritis (RA). Serum from these animals can transfer joint-specific inflammation to normal mice. Fc receptors, interleukin 1, mast cells, and complement are all essential for the development of arthritis after serum transfer. In RA, additional amplifying factors have been identified, including cytokines and intracellular signaling molecules, such as mitogen-activated protein kinases and nuclear factor kappa B, that perpetuate inflammation. Understanding the autoimmune and inflammatory pathways implicated in disease has led to targeted drug development and improved clinical outcomes.
Authors:
Gary S Firestein; Maripat Corr
Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  The Journal of rheumatology. Supplement     Volume:  73     ISSN:  0380-0903     ISO Abbreviation:  J Rheumatol Suppl     Publication Date:  2005 Feb 
Date Detail:
Created Date:  2005-02-04     Completed Date:  2005-04-28     Revised Date:  2005-11-16    
Medline Journal Info:
Nlm Unique ID:  7806058     Medline TA:  J Rheumatol Suppl     Country:  Canada    
Other Details:
Languages:  eng     Pagination:  8-13; discussion 29-30     Citation Subset:  IM    
Affiliation:
Division of Rheumatology, Allergy, and Immunology, Department of Medicine, University of California at San Diego, San Diego, California, USA. gfirestein@ucsd.edu
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Arthritis, Experimental / enzymology,  genetics,  immunology*
Arthritis, Rheumatoid / enzymology,  genetics,  immunology*
Genetic Predisposition to Disease
Glucose-6-Phosphate Isomerase / immunology
Humans
Immunization, Passive
Mice
Mice, Transgenic
Chemical
Reg. No./Substance:
EC 5.3.1.9/Glucose-6-Phosphate Isomerase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Is there a need for new therapies for rheumatoid arthritis?
Next Document:  The role of B cells in the pathogenesis of rheumatoid arthritis.