Document Detail


Combined effects of the two reciprocal t(4;11) fusion proteins MLL.AF4 and AF4.MLL confer resistance to apoptosis, cell cycling capacity and growth transformation.
MedLine Citation:
PMID:  17130830     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The reciprocal chromosomal translocation t(4;11) is correlated with infant, childhood, adult and therapy-related high-risk acute leukemia. Here, we investigated the biological effects of MLL.AF4, AF4.MLL or the combination of both reciprocal fusion proteins in a conditional in vitro cell culture model system. Several parameters like cell growth, cell cycling capacity, apoptotic behavior and growth transformation were investigated under physiological and stress conditions. Co-transfected cells displayed the highest resistance against apoptotic triggers, cell cycling capacity and loss-of-contact inhibition. These analyses were complemented by gene expression profiling experiments and specific gene signatures were established for each of the three cell lines. Interestingly, co-transfected cells strongly upregulate the homeobox gene Nanog. In combination with Oct4, the Nanog homeoprotein is steering maintenance of pluripotency and self-renewal in embryonic stem cells. Transcription of Nanog and other stem cell factors, like Oct4 and Bmi1, was verified in biopsy material of t(4;11) patient cells which express both reciprocal t(4;11) fusion genes. In conclusion, the presence of both reciprocal MLL fusion proteins confers biological properties known from t(4;11) leukemia, suggesting that each of the two fusion proteins contribute specific properties and, in combination, also synergistic effects to the leukemic phenotype.
Authors:
A Gaussmann; T Wenger; I Eberle; A Bursen; S Bracharz; I Herr; T Dingermann; R Marschalek
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2006-11-27
Journal Detail:
Title:  Oncogene     Volume:  26     ISSN:  0950-9232     ISO Abbreviation:  Oncogene     Publication Date:  2007 May 
Date Detail:
Created Date:  2007-05-17     Completed Date:  2007-06-18     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8711562     Medline TA:  Oncogene     Country:  England    
Other Details:
Languages:  eng     Pagination:  3352-63     Citation Subset:  IM    
Affiliation:
Institute of Pharmaceutical Biology/ZAFES, JWG-University Frankfurt, Biocenter, Frankfurt/Main, Germany.
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MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis*
Cell Cycle*
Cells, Cultured
Chromosomes, Human, Pair 4 / genetics*
Cyclin-Dependent Kinase Inhibitor Proteins / genetics
Gene Expression Profiling
Humans
Mice
Mutation / genetics
Myeloid-Lymphoid Leukemia Protein / genetics*,  metabolism*
Oncogene Proteins, Fusion / genetics*,  metabolism*
Polymerase Chain Reaction
Transcription Factors / genetics
Chemical
Reg. No./Substance:
0/Cyclin-Dependent Kinase Inhibitor Proteins; 0/MLL-AF4 fusion protein, human; 0/Oncogene Proteins, Fusion; 0/Transcription Factors; 149025-06-9/Myeloid-Lymphoid Leukemia Protein

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