| Clinico-biochemical, immunological, and morphological assessment of myocardial inflammatory reactions in myocardial necroses. | |
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MedLine Citation:
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PMID: 1212869 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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In 328 patients with myocardial infarction, changes were followed occurring in the levels of haptoglobin (Hp) and chloride-soluble mucoprotein (CMP), heterogeneity of mucoprotein, and titres of anticardial antibodies. From the 2nd-3rd days after infarction, the levels of Hp, CMP, and 3rd and 4th mucoprotein fractions increased and remained elevated for 4 weeks; then the levels gradually sank, and normalized in the 7th-8th weeks after infarction. The anticardial antibody titres remained high from the 3rd to the 6th weeks. In 37 patients who died, the clinico-biochemical, immunobiological, and morphological findings were compared. It was found that assessments of the Hp and CMP levels as well as of mucoprotein heterogeneity make it possible to appraise the intensity of inflammatory reaction in myocardial necroses and thus facilitate the choice of differentiated antiinflammatory treatment of patients with myocardial infarction. |
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Authors:
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S V Shestakov; V N Fatenkov; B L Movshovich; T A Naddachina; V M Rusakov |
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Publication Detail:
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Type: Journal Article |
Journal Detail:
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Title: Cor et vasa Volume: 17 ISSN: 0010-8650 ISO Abbreviation: Cor Vasa Publication Date: 1975 |
Date Detail:
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Created Date: 1976-04-15 Completed Date: 1976-04-15 Revised Date: 2004-11-17 |
Medline Journal Info:
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Nlm Unique ID: 0372614 Medline TA: Cor Vasa Country: CZECHOSLOVAKIA |
Other Details:
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Languages: eng Pagination: 254-61 Citation Subset: IM |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Antibodies
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analysis Glycoproteins / blood Haptoglobins / blood Humans Mucoproteins / blood Myocardial Infarction / blood, immunology, pathology* Myocardium / immunology, pathology* Necrosis |
| Chemical | |
Reg. No./Substance:
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0/Antibodies; 0/Glycoproteins; 0/Haptoglobins; 0/Mucoproteins |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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