Document Detail


Clinical features of the congenital vitreoretinopathies.
MedLine Citation:
PMID:  18309337     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The inherited vitreoretinal degenerations or vitreoretinopathies are characterized by congenital and acquired disorders of the eye including early onset cataract, anomalies of the vitreous manifesting as optically empty vitreous, course fibrils, and membranes, and retinal detachment. These diseases include Stickler syndrome types I (STL1) and II (STL2), usually caused by mutations in COL2A1 and COL11A1 respectively. Wagner syndrome (WGN1) is associated with mutations in versican (CSPG2) and snowflake vitreoretinal degeneration (SVD) with a mutation in a potassium channel (KCNJ13). The cataract is often cortical and may be wedge-shaped, but does not distinguish between the different syndromes. The congenital vitreous defect is usually characterized as fibrillar degeneration (STL2, WGN1, and SVD) or as a vestigial membrane just behind the lens (STL1). Peripheral chorioretinal atrophy with nyctalopia is prominent in WGN1. Intraretinal crystals may be visible in the periphery using a contact lens in SVD and corneal guttae, a flat appearance to the optic nerve head and mild atrophy of the peripheral retinal pigment epithelium are also common features. Other vitreoretinal degenerations including a number of chondrodysplasias in addition to STL1 and STL2, enhanced S-cone syndrome caused by mutations in NR2E3, and autosomal dominant vitreoretinochoroidopathy caused by mutations in VMD2 are discussed. Patients with unexplained early onset cataract or retinal detachment should be carefully evaluated for vitreoretinal degeneration. Theses diseases share overlapping clinical features with common complex traits affecting the eye (myopia, corneal endothelial dystrophy, lattice degeneration), and may provide insight into the mechanisms of common eye diseases.
Authors:
A O Edwards
Related Documents :
20813567 - Panayiotopoulos syndrome: probable genetic origin, but not in scn1a.
17450177 - Werner and hutchinson-gilford progeria syndromes: mechanistic basis of human progeroid ...
20734427 - Clericuzio-type poikiloderma with neutropenia syndrome in three sibs with mutations in ...
15245427 - A phenotype resembling the clouston syndrome with deafness is associated with a novel m...
14697157 - Management of paraneoplastic syndromes in lung cancer.
3688367 - Cutaneous microthrombi: a histologic clue to the diagnosis of hypereosinophilic syndrome.
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review     Date:  2008-02-29
Journal Detail:
Title:  Eye (London, England)     Volume:  22     ISSN:  1476-5454     ISO Abbreviation:  Eye (Lond)     Publication Date:  2008 Oct 
Date Detail:
Created Date:  2008-10-16     Completed Date:  2009-05-18     Revised Date:  2009-11-03    
Medline Journal Info:
Nlm Unique ID:  8703986     Medline TA:  Eye (Lond)     Country:  England    
Other Details:
Languages:  eng     Pagination:  1233-42     Citation Subset:  IM    
Affiliation:
Department of Ophthalmology, Mayo Clinic, Rochester, MN 55905, USA. edwardslab@mayo.edu
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Cataract / etiology
Collagen Type II / genetics
DNA Mutational Analysis
Humans
Mutation / genetics
Retinal Degeneration / genetics
Retinal Detachment / etiology
Retinal Diseases / genetics*
Retinitis Pigmentosa / genetics
Syndrome
Vitreous Body / abnormalities*
Grant Support
ID/Acronym/Agency:
EY014467/EY/NEI NIH HHS
Chemical
Reg. No./Substance:
0/COL2A1 protein, human; 0/Collagen Type II

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Our experience using primary oral antibiotics in the management of orbital cellulitis in a tertiary ...
Next Document:  The influence of pre-mRNA splicing on phenotypic modification in Stickler's syndrome and other type ...