Document Detail


Cigarette smoke induces rapid changes in gene expression in pulmonary arteries.
MedLine Citation:
PMID:  12379773     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The pathogenesis of cigarette smoke-induced pulmonary hypertension is not well characterized. We used RT-PCR to examine gene expression of nitric oxide synthase 2 (NOS-2), nitric oxide synthase 3 (NOS-3), endothelin, and vascular endothelial growth factor (VEGF) and its flk-1 receptor (VEGF-R) in main pulmonary arteries and in intraparenchymal arteries microdissected from alcohol-fixed paraffin blocks. The main pulmonary artery and intraparenchymal vessels responded in a similar fashion, with up-regulation of endothelin, VEGF, and VEGF-R gene expression evident by 2 hours after smoke exposure. Up-regulation of gene expression was still present at 24 hours after exposure, and at this time there was also a small increase in NOS-2. As a comparison, we examined the trachea and microdissected intraparenchymal airways and found up-regulation of endothelin and NOS-2 at 2 hours and additional up-regulation of NOS-3 at 24 hours. These findings suggest that the pulmonary vasculature very rapidly responds to cigarette smoke with up-regulation of mediators that control vascular cell proliferation and vascular constriction. These changes support the idea that pulmonary hypertension in cigarette smokers reflects a direct effect of smoke on the vasculature. The pattern of response in the vessels is distinctly different from that in the airways.
Authors:
Joanne L Wright; Hsin Tai; Jin Dai; Andrew Churg
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Laboratory investigation; a journal of technical methods and pathology     Volume:  82     ISSN:  0023-6837     ISO Abbreviation:  Lab. Invest.     Publication Date:  2002 Oct 
Date Detail:
Created Date:  2002-10-15     Completed Date:  2002-11-19     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0376617     Medline TA:  Lab Invest     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1391-8     Citation Subset:  IM    
Affiliation:
Department of Pathology, University of British Columbia, Vancouver, British Columbia, Canada. jlwright@interchange.ubc.ca
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MeSH Terms
Descriptor/Qualifier:
Animals
Disease Models, Animal
Gene Expression Regulation*
Hypertension, Pulmonary / etiology*
Muscle, Smooth, Vascular / pathology
Pulmonary Artery / pathology*
Rats
Rats, Sprague-Dawley
Reverse Transcriptase Polymerase Chain Reaction
Smoke / adverse effects*
Smoking / adverse effects*
Chemical
Reg. No./Substance:
0/Smoke

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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