| Chronic intestinal ischaemia: diagnosis. | |
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MedLine Citation:
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PMID: 18005164 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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Chronic intestinal ischaemia is a relatively rare but very important clinical entity, which is caused by a reduction in the splanchnic blood flow, most often because of atherosclerosis. Intestinal angina is postprandial abdominal pain developing when the genuine and collateral vessels no longer are able to accommodate the postprandial increasing demand from the gastrointestinal tract and the liver. In addition, the clinical picture very often includes sitophobia and weight loss. In daily clinical practice, conventional angiography is considered as the gold standard, but ultra sonography, computerized tomography and magnetic resonance angiography are gaining momentum when investigating for chronic intestinal ischaemia. These methods depend on imaging of the stenotic vessels not taking into consideration the possibility of sufficient splanchnic perfusion in spite of severely stenotic or occluded vessels. Only a few papers address the physiological consequence of stenotic or occluded vessels - the lack of postprandial increase in splanchnic blood flow. |
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Authors:
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Helle D Zacho; Jan Abrahamsen |
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Publication Detail:
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Type: Journal Article; Review Date: 2007-11-12 |
Journal Detail:
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Title: Clinical physiology and functional imaging Volume: 28 ISSN: 1475-0961 ISO Abbreviation: Clin Physiol Funct Imaging Publication Date: 2008 Mar |
Date Detail:
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Created Date: 2008-02-11 Completed Date: 2008-06-03 Revised Date: - |
Medline Journal Info:
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Nlm Unique ID: 101137604 Medline TA: Clin Physiol Funct Imaging Country: England |
Other Details:
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Languages: eng Pagination: 71-5 Citation Subset: IM |
Affiliation:
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Department of Clinical Physiology, Viborg Hospital, Viborg, Denmark. frusoerensen@hotmail.com |
Export Citation:
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| MeSH Terms | |
Descriptor/Qualifier:
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Chronic Disease Diagnostic Imaging* Humans Intestines / blood supply* Ischemia / diagnosis* Postprandial Period Splanchnic Circulation / physiology* |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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