Document Detail


Chronic changes in blood flow alter endothelium-dependent responses in autogenous vein grafts in dogs.
MedLine Citation:
PMID:  7526008     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
PURPOSE: Experiments were designed to determine the effects of blood flow on endothelium-dependent relaxations in canine vein grafts. METHODS: Blood flow through reversed femoral vein grafts was either increased by a distal arteriovenous fistula (increased flow), unmanipulated (normal flow), or reduced by a proximal adjustable clamp (reduced flow). Six weeks after implantation, blood flow through the graft was measured. Rings cut from grafts were suspended for the measurement of isometric force in organ chambers to determine endothelial function. RESULTS: Blood flow was significantly greater in grafts with a distal fistula compared to grafts with normal or decreased flow. Endothelium-dependent relaxations to acetylcholine were absent in all grafts. Endothelium-dependent relaxations to adenosine diphosphate, thrombin, and the calcium ionophore A23187 were less in grafts with reduced flow compared with grafts with increased flow. Relaxations to these agents in grafts with increased flow were reduced by an analog of L-arginine. Neointimal hyperplasia was increased in grafts with reduced flow. CONCLUSIONS: These data demonstrate that chronic diminution of blood flow decreases receptor-mediated release of endothelium-derived relaxing factors and increases neointimal hyperplasia in canine vein grafts. The production of endothelium-derived relaxing factors, one of which is nitric oxide, may influence the development of myointimal hyperplasia in vein grafts.
Authors:
R A Cambria; R C Lowell; P Gloviczki; V M Miller
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Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Journal of vascular surgery : official publication, the Society for Vascular Surgery [and] International Society for Cardiovascular Surgery, North American Chapter     Volume:  20     ISSN:  0741-5214     ISO Abbreviation:  J. Vasc. Surg.     Publication Date:  1994 Nov 
Date Detail:
Created Date:  1994-12-27     Completed Date:  1994-12-27     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  8407742     Medline TA:  J Vasc Surg     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  765-73     Citation Subset:  IM    
Affiliation:
Department of Surgery, Mayo Clinic and Foundation, Rochester, MN 55905.
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MeSH Terms
Descriptor/Qualifier:
Acetylcholine / pharmacology
Adenosine Diphosphate / pharmacology
Adrenergic alpha-Agonists / pharmacology
Animals
Arginine / analogs & derivatives,  pharmacology
Arteriovenous Shunt, Surgical
Blood Flow Velocity
Calcimycin / pharmacology
Constriction
Dinoprost / pharmacology
Dogs
Endothelins / pharmacology
Endothelium, Vascular / drug effects,  pathology,  physiopathology*
Femoral Artery / drug effects,  pathology,  physiopathology*,  surgery*
Femoral Vein / drug effects,  pathology,  physiopathology*,  transplantation*
Hyperplasia
Male
Models, Biological
Neovascularization, Pathologic / physiopathology
Nitric Oxide / antagonists & inhibitors
Quinoxalines / pharmacology
Thrombin / pharmacology
Tunica Intima / drug effects,  pathology,  physiopathology*
Vascular Patency
omega-N-Methylarginine
Grant Support
ID/Acronym/Agency:
HL-42614/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Adrenergic alpha-Agonists; 0/Endothelins; 0/Quinoxalines; 10102-43-9/Nitric Oxide; 17035-90-4/omega-N-Methylarginine; 51-84-3/Acetylcholine; 52665-69-7/Calcimycin; 551-11-1/Dinoprost; 58-64-0/Adenosine Diphosphate; 59803-98-4/brimonidine; 74-79-3/Arginine; EC 3.4.21.5/Thrombin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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