Document Detail


Cholangiocyte bile salt transporters in cholesterol gallstone-susceptible and resistant inbred mouse strains.
MedLine Citation:
PMID:  18717763     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND AND AIM: We investigated the dietary and gender influences on the expression and functionality of cholangiocyte bile salt transporters and development of biliary hyperplasia in cholesterol gallstone-susceptible C57L/J and resistant AKR/J mice.
METHODS: C57L and AKR mice were fed chow, a lithogenic diet, or a cholic acid-containing diet for 14 days. Expression of cholangiocyte bile salt transporter proteins ASBT (SLC10A2), ILBP (FABP6), and MRP3 (ABCC3) were studied by Western blot analysis. Taurocholate uptake studies were performed using microperfusion of isolated bile duct units. The pre- and post-perfusion taurocholate concentrations were analyzed by high performance liquid chromatography. Biliary proliferation in liver sections was scored.
RESULTS: The lithogenic diet induced ductular proliferation in C57L mice. On chow, SLC10A2 and ABCC3 were overexpressed in male and female C57L compared to AKR mice. A lithogenic diet reduced the expressions of FABP6 in both male and female C57L mice, SLC10A2 in female C57L mice, and ABCC3 in male C57L mice. These alterations in transporter expressions were not associated with changes in taurocholate uptake. The lithogenic diet induced biliary hyperplasia and reduced bile salt transporter expressions in C57L mice.
CONCLUSIONS: Although bile salt uptake was not increased in the bile duct unit, we speculate that the biliary hyperplasia on the lithogenic diet may lead to an increase in intrahepatic bile salt recycling during cholesterol cholelithogenesis.
Authors:
Julia J Liu; Jonathan N Glickman; Anatoliy I Masyuk; Nicholas F Larusso
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Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2008-08-20
Journal Detail:
Title:  Journal of gastroenterology and hepatology     Volume:  23     ISSN:  1440-1746     ISO Abbreviation:  J. Gastroenterol. Hepatol.     Publication Date:  2008 Oct 
Date Detail:
Created Date:  2009-01-05     Completed Date:  2009-04-09     Revised Date:  2011-11-01    
Medline Journal Info:
Nlm Unique ID:  8607909     Medline TA:  J Gastroenterol Hepatol     Country:  Australia    
Other Details:
Languages:  eng     Pagination:  1596-602     Citation Subset:  IM    
Affiliation:
Division of Gastroenterology, Department of Medicine, University of Alberta Hospital, Edmonton, Alberta, Canada. julia.liu@ualberta.ca
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MeSH Terms
Descriptor/Qualifier:
Animals
Bile Ducts / metabolism*,  pathology
Cell Proliferation
Cholesterol, Dietary
Disease Models, Animal
Disease Susceptibility
Fatty Acid-Binding Proteins / metabolism
Female
Gallstones / etiology,  metabolism*,  pathology
Gastrointestinal Hormones / metabolism
Hyperplasia
Male
Membrane Transport Proteins / metabolism*
Mice
Mice, Inbred AKR
Mice, Inbred C57BL
Multidrug Resistance-Associated Proteins / metabolism
Organic Anion Transporters, Sodium-Dependent / metabolism
Sex Factors
Species Specificity
Symporters / metabolism
Taurocholic Acid / metabolism*
Grant Support
ID/Acronym/Agency:
DK 24031/DK/NIDDK NIH HHS; DK 36588/DK/NIDDK NIH HHS; DK 52911/DK/NIDDK NIH HHS; DK 57993/DK/NIDDK NIH HHS; R01 DK024031-31/DK/NIDDK NIH HHS; R01 DK057993-07/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Cholesterol, Dietary; 0/Fatty Acid-Binding Proteins; 0/Gastrointestinal Hormones; 0/Membrane Transport Proteins; 0/Multidrug Resistance-Associated Proteins; 0/Organic Anion Transporters, Sodium-Dependent; 0/Symporters; 0/multidrug resistance-associated protein 3; 117849-44-2/fatty acid-binding protein 6; 145420-23-1/sodium-bile acid cotransporter; 81-24-3/Taurocholic Acid

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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