Document Detail


Chk1 activation and the nuclear/cytoplasmic ratio.
MedLine Citation:
PMID:  15296709     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
ATR and Chk1 are important components of a cell cycle checkpoint pathway. In this issue of Developmental Cell, Conn et al. shed a novel light on the molecular mechanism of Chk1 activation and raise the possibility of a developmental checkpoint that regulates Chk1 in response to the nuclear/cytoplasmic ratio.
Authors:
Jennifer Pogoriler; Wei Du
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Publication Detail:
Type:  Comment; Journal Article    
Journal Detail:
Title:  Developmental cell     Volume:  7     ISSN:  1534-5807     ISO Abbreviation:  Dev. Cell     Publication Date:  2004 Aug 
Date Detail:
Created Date:  2004-08-06     Completed Date:  2004-09-07     Revised Date:  2011-11-02    
Medline Journal Info:
Nlm Unique ID:  101120028     Medline TA:  Dev Cell     Country:  United States    
Other Details:
Languages:  eng     Pagination:  147-8     Citation Subset:  IM    
Affiliation:
Ben May Institute for Cancer Research, Committee on Cancer Biology, University of Chicago, 924 East 57th Street, IL 60637 USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Cycle
Cell Cycle Proteins / metabolism*
Cell Nucleus / metabolism*
Cytoplasm / metabolism*
Enzyme Activation
Gene Expression Regulation, Developmental*
Models, Biological
Protein Kinases / metabolism*
Protein-Serine-Threonine Kinases / metabolism*
cdc25 Phosphatases / metabolism
Chemical
Reg. No./Substance:
0/Cell Cycle Proteins; EC 2.7.-/Protein Kinases; EC 2.7.11.1/Checkpoint kinase 1; EC 2.7.11.1/Protein-Serine-Threonine Kinases; EC 3.1.3.48/cdc25 Phosphatases
Comments/Corrections
Comment On:
Dev Cell. 2004 Aug;7(2):275-81   [PMID:  15296723 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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