Document Detail


Characterization of a sialic acid- and P-selectin glycoprotein ligand-1-independent adhesin activity in the granulocytotropic bacterium Anaplasma phagocytophilum.
MedLine Citation:
PMID:  16869829     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Anaplasma phagocytophilum, the aetiologic agent of human granulocytic anaplasmosis, is an obligate intracellular bacterium that colonizes neutrophils and neutrophil precursors. The granulocytotropic bacterium uses multiple adhesins that cooperatively bind to the N-terminal region of P-selectin glycoprotein ligand-1 (PSGL-1) and to sialyl Lewis x (sLe(x)) expressed on myeloid cell surfaces. Recognition of sLe(x) occurs through interactions with alpha2,3-sialic acid and alpha1,3-fucose. It is unknown whether other bacteria-host cell interactions are involved. In this study, we have enriched for A. phagocytophilum organisms that do not rely on sialic acid for cellular adhesion and entry by maintaining strain NCH-1 in HL-60 cells that are severely undersialylated. The selected bacteria, termed NCH-1A, also exhibit lessened dependencies on PSGL-1 and alpha1,3-fucose. Optimal adhesion and invasion by NCH-1A require interactions with the known determinants (sialic acid, PSGL-1 and alpha1,3-fucose), but none of them is absolutely necessary. NCH-1A binding to sLe(x)-modified PSGL-1 requires recognition of the known determinants in the same manners as other A. phagocytophilum strains. These data suggest that A. phagocytophilum expresses a separate adhesin from those targeting sialic acid, alpha1,3-fucose and the N-terminal region of PSGL-1. We propose that NCH-1A upregulates expression of this adhesin.
Authors:
Dexter V Reneer; Sarah A Kearns; Tadayuki Yago; Jonathan Sims; Richard D Cummings; Rodger P McEver; Jason A Carlyon
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2006-07-26
Journal Detail:
Title:  Cellular microbiology     Volume:  8     ISSN:  1462-5814     ISO Abbreviation:  Cell. Microbiol.     Publication Date:  2006 Dec 
Date Detail:
Created Date:  2006-11-09     Completed Date:  2007-01-25     Revised Date:  2007-12-03    
Medline Journal Info:
Nlm Unique ID:  100883691     Medline TA:  Cell Microbiol     Country:  England    
Other Details:
Languages:  eng     Pagination:  1972-84     Citation Subset:  IM    
Affiliation:
Department of Microbiology, Immunology and Molecular Genetics, University of Kentucky College of Medicine, Lexington, KY, USA.
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MeSH Terms
Descriptor/Qualifier:
Adhesins, Bacterial / chemistry,  metabolism*
Anaplasma phagocytophilum / metabolism*,  pathogenicity
Bacterial Adhesion*
Fucose / metabolism
HL-60 Cells
Humans
Membrane Glycoproteins / metabolism
N-Acetylneuraminic Acid / metabolism
Grant Support
ID/Acronym/Agency:
AI065631/AI/NIAID NIH HHS; AI48075/AI/NIAID NIH HHS; DK065039/DK/NIDDK NIH HHS; HL65631/HL/NHLBI NIH HHS; P20 RR020171/RR/NCRR NIH HHS
Chemical
Reg. No./Substance:
0/Adhesins, Bacterial; 0/Membrane Glycoproteins; 0/P-selectin ligand protein; 131-48-6/N-Acetylneuraminic Acid; 3713-31-3/Fucose

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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