Document Detail


Changes in transcription profile and cytoskeleton morphology in pelvic ligament fibroblasts in response to stretch: the effects of estradiol and levormeloxifene.
MedLine Citation:
PMID:  18184756     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Failure of ligamentous support of the genital tract to resist intra-abdominal pressure is a plausible underlying mechanism for the development of pelvic organ prolapse, but the nature of the molecular response of pelvic tissue support remains unknown. We hypothesized that the expression of genes coding for proteins involved in maintaining the cellular and extracellular integrity would be altered as a result of mechanical stretch. Therefore, cDNA microarrays were used to examine the difference in transcriptional profile in RNA of primary culture fibroblasts subjected to mechanical stretch and those that remained static. Out of 34 mechano-responsive genes identified (P < 0.05), four were coding for regulation of actin cytoskeleton remodelling, and its interaction with the extracellular matrix proteins; these are phosphatidyl inositol-4-phosphate 5-kinase (PIP5K1C), the human signal-induced proliferation associated gene-1 (SIPA-1), TNFRSF1A-associated via death domain (TRADD) and deoxyribonuclease 1-like 1 (DNase 1-L1). The transcriptosomal changes led us to investigate the phenotypic consequences of stretch, levormeloxifene and estradiol (E(2)) on the cytoskeleton of cultured fibroblasts. The percentage of cells with abnormal F-actin configuration was significantly higher in fibroblasts subjected to stretch compared with the static model (P < 0.0001). Levormeloxifene caused similar significant alterations in actin morphology of the static fibroblasts. The use of E(2) did not reverse the process or protect the cells from the effect of stretch, but significantly increased the rate of fibroblast proliferation, suggestive of a role in healing process. Mechanical stretch and/or levormeloxifene disturb the fibroblasts ability to maintain the cytoskeleton architecture and we speculate that they may disrupt ligamentous integrity and result in clinical prolapse.
Authors:
Ayman A A Ewies; Mona Elshafie; Jin Li; Adrian Stanley; John Thompson; Jerry Styles; Ian White; Farook Al-Azzawi
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2008-01-09
Journal Detail:
Title:  Molecular human reproduction     Volume:  14     ISSN:  1460-2407     ISO Abbreviation:  Mol. Hum. Reprod.     Publication Date:  2008 Feb 
Date Detail:
Created Date:  2008-02-19     Completed Date:  2008-07-15     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9513710     Medline TA:  Mol Hum Reprod     Country:  England    
Other Details:
Languages:  eng     Pagination:  127-35     Citation Subset:  IM    
Affiliation:
Department of Gynaecology, The Ipswich Hospital, Ipswich, UK.
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MeSH Terms
Descriptor/Qualifier:
Actins / genetics,  metabolism
Blotting, Western
Cell Proliferation / drug effects
Cells, Cultured
Estradiol / pharmacology*
Fibroblasts / cytology,  drug effects*,  metabolism
Gene Expression Profiling*
Humans
Ligaments / cytology*
Oligonucleotide Array Sequence Analysis
Pelvis
Pyrrolidines / pharmacology*
Stress, Mechanical
Transcription, Genetic / drug effects
Tubulin / genetics,  metabolism
Chemical
Reg. No./Substance:
0/Actins; 0/Pyrrolidines; 0/Tubulin; 50-28-2/Estradiol; 78994-23-7/levormeloxifene

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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