Document Detail


Changes in the pattern of microsomal fatty acids in rat liver after thermal injury and therapeutic intervention.
MedLine Citation:
PMID:  3370512     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Despite substantial progress in handling the acute phase after thermal injury, severely burned patients still succumb to systemic sepsis as a consequence of a compromised defence system. It is likely that autotoxic mechanisms play an important role in the aetiology of the impaired host defence. One of the primary target systems of autotoxic cell damage is the liver. In the present work oxidative alterations in the microsomal compartment of liver cells have been investigated. It was found that thermal burns are associated with extensive oxidation of polyunsaturated fatty acids which can be antagonized by antioxidants such as silibinin.
Authors:
K H Schmidt; U Müller; W Hörer; R Braatz
Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Burns, including thermal injury     Volume:  14     ISSN:  0305-4179     ISO Abbreviation:  Burns Incl Therm Inj     Publication Date:  1988 Feb 
Date Detail:
Created Date:  1988-06-30     Completed Date:  1988-06-30     Revised Date:  2003-11-14    
Medline Journal Info:
Nlm Unique ID:  7512054     Medline TA:  Burns Incl Therm Inj     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  25-30     Citation Subset:  IM    
Affiliation:
Department of Surgery, University of Tuebingen, FR Germany.
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MeSH Terms
Descriptor/Qualifier:
Animals
Antioxidants / therapeutic use*
Burns / complications*,  drug therapy
Fatty Acids / metabolism*
Fatty Acids, Unsaturated / metabolism
Male
Microsomes, Liver / metabolism*
Rats
Rats, Inbred Strains
Silymarin / therapeutic use
Chemical
Reg. No./Substance:
0/Antioxidants; 0/Fatty Acids; 0/Fatty Acids, Unsaturated; 0/Silymarin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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