Document Detail


Changes in intracellular electrolyte concentrations during apoptosis induced by UV irradiation of human myeloblastic cells.
MedLine Citation:
PMID:  16162654     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Decreases in the intracellular concentrations of both K(+) and Cl(-) have been implicated in playing a major role in the progression of apoptosis, but little is known about the temporal relationship between decreases in electrolyte concentration and the key events in apoptosis, and there is no information about how such decreases affect different intracellular compartments. Electron probe X-ray microanalysis was used to determine changes in element concentrations (Na, P, Cl, and K) in nucleus, cytoplasm, and mitochondria in U937 cells undergoing UV-induced apoptosis. In all compartments, the initial stages of apoptosis were characterized by decreases in [K] and [Cl]. The largest decreases in these elements were in the mitochondria and occurred before the release of cytochrome c. Initial decreases in [K] and [Cl] also preceded apoptotic changes in the nucleus. In the later stages of apoptosis, the [K] continued to decrease, whereas that of Cl began to increase toward control levels and was accompanied by an increase in [Na]. In the nucleus, these increases coincided with poly(ADP-ribose) polymerase cleavage, chromatin condensation, and DNA laddering. The cytoplasm was the compartment least affected and the pattern of change of Cl was similar to those in other compartments, but the decrease in [K] was not significant until after active caspase-3 was detected. Our results support the concept that normotonic cell shrinkage occurs early in apoptosis, and demonstrate that changes in the intracellular concentrations of K and Cl precede apoptotic changes in the cell compartments studied.
Authors:
F Arrebola; E Fernández-Segura; A Campos; P V Crespo; J N Skepper; A Warley
Related Documents :
18281494 - Acetyl-keto-beta-boswellic acid induces apoptosis through a death receptor 5-mediated p...
18758954 - Parp and cell death or protection in rat primary astroglial cell cultures under lps/ifn...
19913524 - Apoptosis induction in human leukemic cells by a novel protein bengalin, isolated from ...
20814064 - Superoxide is a potential culprit of caspase-3 dependent endothelial cell death induced...
19414804 - Potent phagocytic activity with impaired antigen presentation identifying lipopolysacch...
21413004 - Expression of il-10-triggered stat3-dependent il-4rα is required for induction of argi...
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2005-09-14
Journal Detail:
Title:  American journal of physiology. Cell physiology     Volume:  290     ISSN:  0363-6143     ISO Abbreviation:  Am. J. Physiol., Cell Physiol.     Publication Date:  2006 Feb 
Date Detail:
Created Date:  2006-01-11     Completed Date:  2006-03-10     Revised Date:  2007-08-13    
Medline Journal Info:
Nlm Unique ID:  100901225     Medline TA:  Am J Physiol Cell Physiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  C638-49     Citation Subset:  IM    
Affiliation:
Electron Microscopy Unit, King's College London, Department of Ophthalmology, The Rayne Institute, St. Thomas' Hospital, UK.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis / radiation effects*
Cell Line, Tumor
Cell Nucleus / chemistry*,  radiation effects
Chlorine / metabolism
Cytoplasm / chemistry*
Electrolytes*
Electron Probe Microanalysis
Humans
Lymphoma
Mitochondria / chemistry*,  radiation effects
Phosphorus / metabolism
Potassium / metabolism
Sodium / metabolism
Ultraviolet Rays
Grant Support
ID/Acronym/Agency:
//Wellcome Trust
Chemical
Reg. No./Substance:
0/Electrolytes; 7440-09-7/Potassium; 7440-23-5/Sodium; 7723-14-0/Phosphorus; 7782-50-5/Chlorine

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Regulation of ocular lens development by Smad-interacting protein 1 involving Foxe3 activation.
Next Document:  Heat shock-induced attenuation of hydroxyl radical generation and mitochondrial aconitase activity i...