Document Detail


Cerebrovascular damage in late-life depression is associated with structural and functional abnormalities of subcutaneous small arteries.
MedLine Citation:
PMID:  20713917     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Late-life depression is increasingly viewed as a vascular illness because of patients exhibiting characteristic white matter brain lesions and in vivo large artery endothelial dysfunction. However, the "vascular depression" hypothesis pertains to the microvasculature, and this circulation has not been studied in this context. Our objective was to examine structure and function of small subcutaneous arteries in patients with late-life depression. Thus, 16 patients aged 71.8±4.0 years with late-life depression were compared with 15 control participants aged 72.1±5.9 years. There were similar cardiovascular profiles between the 2 groups. All of the participants underwent MRI brain scans and subcutaneous gluteal fat biopsy from which small arteries were isolated and studied using pressure myography. Cerebral microvascular damage in depressed patients was confirmed by assessment of basal ganglia Virchow-Robin space scores (depressed patients 3.9±1.7 versus controls: 2.5±1.6; P=0.01). Contractility to norepinephrine was equivalent in both groups, but relaxation of the small arteries to acetylcholine was significantly reduced in depressed patients (84.0±4.0%) compared with control participants (96.0±1.4%; P=0.012). This difference in arterial relaxation was reduced but not entirely eliminated when NO synthase was inhibited. Depressed patients also exhibited hypertrophic wall growth with an increase in medial cross-sectional area (P=0.035, multiple ANOVA and wall thickness; P=0.04, multiple ANOVA). In conclusion, despite similar cardiovascular profiles, depressed patients with cerebral microvascular damage show abnormalities of subcutaneous small artery structure and function.
Authors:
Adam S Greenstein; Raghupathy Paranthaman; Alistair Burns; Alan Jackson; Rayaz A Malik; Robert C Baldwin; Anthony M Heagerty
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Publication Detail:
Type:  In Vitro; Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-08-16
Journal Detail:
Title:  Hypertension     Volume:  56     ISSN:  1524-4563     ISO Abbreviation:  Hypertension     Publication Date:  2010 Oct 
Date Detail:
Created Date:  2010-09-16     Completed Date:  2010-10-29     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  7906255     Medline TA:  Hypertension     Country:  United States    
Other Details:
Languages:  eng     Pagination:  734-40     Citation Subset:  IM    
Affiliation:
Universityof Manchester, Manchester, United Kingdom.
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MeSH Terms
Descriptor/Qualifier:
Acetylcholine / pharmacology
Aged
Analysis of Variance
Arteries / abnormalities*,  physiopathology
Brain / pathology,  physiopathology
Cerebrovascular Disorders / complications,  pathology,  physiopathology*
Depressive Disorder / complications*
Dose-Response Relationship, Drug
Female
Humans
Magnetic Resonance Imaging
Male
Myography
Norepinephrine / pharmacology
Subcutaneous Tissue / blood supply*
Vasoconstriction / drug effects
Vasodilator Agents / pharmacology
Chemical
Reg. No./Substance:
0/Vasodilator Agents; 51-41-2/Norepinephrine; 51-84-3/Acetylcholine

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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