Document Detail


Cell growth inhibition, G2/M cell cycle arrest, and apoptosis induced by chloroquine in human breast cancer cell line Bcap-37.
MedLine Citation:
PMID:  19088425     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Chloroquine is an antimalarial drug that has been used in the treatment and prophylaxis of malaria since the 1950s. The present study was undertaken to examine the effects of chloroquine on Bcap-37 human breast cancer cells' growth, cell cycle modulation, apoptosis induction, and associated molecular alterations in vitro. The chloroquine treatment decreased the viability of Bcap-37 cells in a concentration- and time-dependent manner, which correlated with G(2)/M phase cell cycle arrest. The chloroquine-mediated cell cycle arrest was associated with a decrease in protein levels/activity of polo-like kinase 1 (Plk1), phosphorylated cell division cycle 25C (Cdc25C), phosphorylated extracellular signal-regulated kinase 1/2 (ERK1/2), phosphorylated Akt. The chloroquine-treated Bcap-37 cells exhibited a marked decrease in the level of mitochondrial transmembrane potential (DeltaPsim), which was accompanied by the activation of caspase-3 and cleaved poly(ADP-ribose) polymerase (PARP). Exposure of Bcap-37 cells to chloroquine also resulted in the induction of spindle abnormalities. In conclusion, the findings in this study suggested that chloroquine might have potential anticancer efficacy, which could be attributed, in part, to its proliferation inhibition and apoptosis induction of cancer cells through modulation of apoptosis and cell cycle-related proteins expressions, down-regulation of mitochondrial transmembrane potential (DeltaPsim), and induction of spindle abnormalities.
Authors:
Pei-du Jiang; Ying-lan Zhao; Wei Shi; Xiao-qiang Deng; Gang Xie; Yong-qiu Mao; Zheng-guang Li; Yu-zhu Zheng; Sheng-yong Yang; Yu-quan Wei
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2008-12-09
Journal Detail:
Title:  Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology     Volume:  22     ISSN:  1421-9778     ISO Abbreviation:  Cell. Physiol. Biochem.     Publication Date:  2008  
Date Detail:
Created Date:  2008-12-17     Completed Date:  2009-02-10     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9113221     Medline TA:  Cell Physiol Biochem     Country:  Switzerland    
Other Details:
Languages:  eng     Pagination:  431-40     Citation Subset:  IM    
Copyright Information:
Copyright 2008 S. Karger AG, Basel.
Affiliation:
State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, West China Medical School, Sichuan University, Chengdu, Sichuan, China.
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MeSH Terms
Descriptor/Qualifier:
Apoptosis / drug effects*
Breast Neoplasms / enzymology,  pathology*
Caspase 3 / metabolism
Cell Cycle Proteins / metabolism
Cell Division / drug effects*
Cell Line, Tumor
Cell Proliferation / drug effects
Cell Shape / drug effects
Chloroquine / pharmacology*
Drug Screening Assays, Antitumor
Enzyme Activation / drug effects
G2 Phase / drug effects*
Humans
Membrane Potential, Mitochondrial / drug effects
Mitotic Spindle Apparatus / drug effects
Chemical
Reg. No./Substance:
0/Cell Cycle Proteins; 54-05-7/Chloroquine; EC 3.4.22.-/Caspase 3

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