Document Detail


Review: cell cycle aberrations and neurodegeneration.
MedLine Citation:
PMID:  20059701     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The cell cycle is a highly regulated and fundamental cellular process that involves complex feedback regulation of many proteins, and any compromise to its integrity elicits dire consequences for the cell. For example, in neurodegenerative diseases such as Alzheimer disease (AD), evidence for abnormal cell cycle re-entry precedes other hallmarks of disease and as such, implicates cell cycle aberrations in the aetiology of AD. The mechanism(s) for cell cycle re-entry in AD, however, remain unclear. Current theory suggests it to be part of a combination of early events that together elicit the degenerative pathology and cognitive phenotype consistent with the disease. We propose a 'Two-Hit Hypothesis' that highlights the concerted interaction between cell cycle alterations and oxidative stress that combine to produce neurodegeneration. Here, we review the evidence implicating cell cycle mechanisms in AD and how such changes, especially in combination with oxidative stress, would lead to a cascade of events leading to disease. Based on this concept, we propose new opportunities for disease treatment.
Authors:
D J Bonda; V P Bajić; B Spremo-Potparevic; G Casadesus; X Zhu; M A Smith; H-G Lee
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Review     Date:  2010-01-06
Journal Detail:
Title:  Neuropathology and applied neurobiology     Volume:  36     ISSN:  1365-2990     ISO Abbreviation:  Neuropathol. Appl. Neurobiol.     Publication Date:  2010 Apr 
Date Detail:
Created Date:  2010-04-26     Completed Date:  2010-07-19     Revised Date:  2010-12-17    
Medline Journal Info:
Nlm Unique ID:  7609829     Medline TA:  Neuropathol Appl Neurobiol     Country:  England    
Other Details:
Languages:  eng     Pagination:  157-63     Citation Subset:  IM    
Affiliation:
Departments of Pathology, Case Western Reserve University, Cleveland, Ohio 44106, USA.
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MeSH Terms
Descriptor/Qualifier:
Alzheimer Disease / physiopathology,  therapy
Animals
Cell Cycle / physiology*
Humans
Nerve Degeneration / physiopathology,  therapy
Neurodegenerative Diseases / physiopathology*,  therapy
Neurons / physiology
Grant Support
ID/Acronym/Agency:
AG028679/AG/NIA NIH HHS; AG030096/AG/NIA NIH HHS; AG031364/AG/NIA NIH HHS; R01 AG028679-04/AG/NIA NIH HHS; R21 AG030096-02/AG/NIA NIH HHS

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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