Document Detail


Cdk5 suppresses the neuronal cell cycle by disrupting the E2F1-DP1 complex.
MedLine Citation:
PMID:  20392944     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Neurons that reenter a cell cycle after maturation are at increased risk for death, yet the mechanisms by which a normal neuron suppresses the cycle remain mostly unknown. Our laboratory has shown that cyclin-dependent kinase 5 (Cdk5) is a potent cell cycle suppressor, and we report here on the molecular basis of this activity. Cell cycle suppression by Cdk5 requires its binding to the p35 activator protein. The related p39 and p25 proteins cannot serve as substitutes. Unexpectedly, Cdk5 enzymatic activity is not required to perform this function. Rather, the link to cell cycle regulation is made through the formation of a previously unknown complex consisting of the p35-Cdk5 dimer and E2F1. Formation of this complex excludes the E2F1 cofactor, DP1, thus inhibiting E2F1 binding to the promoters of various cell cycle genes. This anti-cell cycle activity is most likely a neuroprotective function of Cdk5.
Authors:
Jie Zhang; Huifang Li; Odessa Yabut; Haley Fitzpatrick; Gabriella D'Arcangelo; Karl Herrup
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural    
Journal Detail:
Title:  The Journal of neuroscience : the official journal of the Society for Neuroscience     Volume:  30     ISSN:  1529-2401     ISO Abbreviation:  J. Neurosci.     Publication Date:  2010 Apr 
Date Detail:
Created Date:  2010-04-15     Completed Date:  2010-05-14     Revised Date:  2012-06-25    
Medline Journal Info:
Nlm Unique ID:  8102140     Medline TA:  J Neurosci     Country:  United States    
Other Details:
Languages:  eng     Pagination:  5219-28     Citation Subset:  IM    
Affiliation:
Department of Cell Biology and Neuroscience, Rutgers, The State University of New Jersey, Piscataway, New Jersey 08854, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Brain / enzymology,  physiology
Cell Cycle / physiology*
Cell Line, Tumor
Cell Nucleus / physiology
Cells, Cultured
Cyclin-Dependent Kinase 5 / deficiency,  genetics,  metabolism*
Cytoplasm / physiology
DNA / metabolism
E2F1 Transcription Factor / metabolism*
Mice
Mice, Inbred C57BL
Mice, Transgenic
Models, Neurological
Nerve Tissue Proteins / metabolism
Neurons / enzymology,  physiology*
Phosphotransferases / metabolism*
Transcription Factor DP1 / metabolism*
Grant Support
ID/Acronym/Agency:
NS20591/NS/NINDS NIH HHS; R01 NS020591-26A2/NS/NINDS NIH HHS
Chemical
Reg. No./Substance:
0/Cdk5r protein, mouse; 0/E2F1 Transcription Factor; 0/E2f1 protein, mouse; 0/Nerve Tissue Proteins; 0/Tfdp1 protein, mouse; 0/Transcription Factor DP1; 0/p25 protein, mouse; 9007-49-2/DNA; EC 2.7.-/Phosphotransferases; EC 2.7.11.22/Cdk5 protein, mouse; EC 2.7.11.22/Cyclin-Dependent Kinase 5

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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