Document Detail


Caveolae are an essential component of the pathway for endothelial cell signaling associated with abrupt reduction of shear stress.
MedLine Citation:
PMID:  18573285     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Abrupt cessation of flow representing the acute loss of shear stress (simulated ischemia) to flow-adapted pulmonary microvascular endothelial cells (PMVEC) leads to reactive oxygen species (ROS) generation that signals for EC proliferation. We evaluated the role of caveolin-1 on this cellular response with mouse PMVEC that were preconditioned for 72 h to laminar flow at 5 dyn/cm(2) followed by stop of flow ("ischemia"). Preconditioning resulted in a 2.7-fold increase in cellular expression of K(ATP) (K(IR) 6.2) channels but no change in expression level of caveolin-1, gp91(phox), or MAP kinases. The initial response to ischemia in wild type cells was cell membrane depolarization that was abolished by gene targeting of K(IR) 6.2. The subsequent response was increased ROS production associated with activation of NADPH oxidase (NOX2) and then phosphorylation of MAP kinases (Erk, JNK). After 24 h of ischemia in wild type cells, the cell proliferation index increased 2.5 fold and the % of cells in S+G(2)/M phases increased 6-fold. This signaling cascade (cell membrane depolarization, ROS production, MAP kinase activation and cell proliferation) was abrogated in caveolin-1 null PMVEC or by treatment of wild type cells with filipin. These studies indicate that caveolin-1 functions as a shear sensor in flow-adapted EC resulting in ROS-mediated cell signaling and endothelial cell proliferation following the abrupt reduction in flow.
Authors:
Tatyana Milovanova; Shampa Chatterjee; Brian J Hawkins; Nankang Hong; Elena M Sorokina; Kris Debolt; Jonni S Moore; Muniswamy Madesh; Aron B Fisher
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2008-05-23
Journal Detail:
Title:  Biochimica et biophysica acta     Volume:  1783     ISSN:  0006-3002     ISO Abbreviation:  Biochim. Biophys. Acta     Publication Date:  2008 Oct 
Date Detail:
Created Date:  2008-09-09     Completed Date:  2008-11-13     Revised Date:  2011-03-10    
Medline Journal Info:
Nlm Unique ID:  0217513     Medline TA:  Biochim Biophys Acta     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  1866-75     Citation Subset:  IM    
Affiliation:
Institute For Environmental Medicine, University of Pennsylvania Medical Center, One John Morgan Building, Philadelphia, PA 19104-6068, USA.
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MeSH Terms
Descriptor/Qualifier:
Adaptation, Physiological
Animals
Caveolae / metabolism*
Caveolin 1 / deficiency,  genetics,  metabolism
Cell Membrane / metabolism
Cells, Cultured
Endothelial Cells / metabolism*
Ischemia / genetics,  metabolism*
Mice
Mice, Inbred C57BL
Mice, Knockout
Mitogen-Activated Protein Kinases / metabolism
Phenotype
Phosphorylation
Reactive Oxygen Species / metabolism
Signal Transduction*
Stress, Mechanical
Grant Support
ID/Acronym/Agency:
HL75587/HL/NHLBI NIH HHS; P50 HL060290-040002/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Caveolin 1; 0/Reactive Oxygen Species; EC 2.7.11.24/Mitogen-Activated Protein Kinases
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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