Document Detail


Cathepsin-B-dependent apoptosis triggered by antithymocyte globulins: a novel mechanism of T-cell depletion.
MedLine Citation:
PMID:  12893746     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Antithymocyte globulins (ATGs), the immunoglobulin G (IgG) fraction of sera from rabbits or horses immunized with human thymocytes or T-cell lines, are used in conditioning regimens for bone marrow transplantation, in the treatment of acute graft-versus-host disease, in the prevention or treatment of acute rejection in organ transplantation, and in severe bone marrow aplasia. In nonhuman primates, ATGs induce rapid, dose-dependent, T-cell depletion in peripheral lymphoid tissues, where apoptotic cells can be demonstrated in T-cell zones. We show here that increasing ATG concentrations in vitro resulted in reduced lymphocyte proliferative responses, associated with a rapid increase in the percentage of apoptotic cells. Apoptosis did not require prior exposure to interleukin-2, nor did it result in CD178/CD95 or tumor necrosis factor/tumor necrosis factor receptor (TNF/TNF-R) interactions; it was therefore clearly different from activation-induced cell death. Cytochrome c release, caspase-9, and caspase-3 activation were not implicated, excluding a direct involvement of the intrinsic mitochondrial pathway. The cysteine protease inhibitor E64d and cathepsin-B-specific inhibitors conferred significant protection, whereas apoptosis was associated with the release of active cathepsin B into the cytosol. These data demonstrate a role for cathepsin B in T-cell apoptosis induced by ATGs at concentrations achieved during clinical use.
Authors:
Marie-Cécile Michallet; Frederic Saltel; Xavier Preville; Monique Flacher; Jean-Pierre Revillard; Laurent Genestier
Related Documents :
9493956 - Cleavage of poly(adp-ribose) polymerase: a sensitive parameter to study cell death.
10502406 - Paclitaxel induces apoptosis in saos-2 cells with cd95l upregulation and bcl-2 phosphor...
14581476 - Selective disruption of lysosomes in hela cells triggers apoptosis mediated by cleavage...
19380466 - Novel subtilase cytotoxin produced by shiga-toxigenic escherichia coli induces apoptosi...
18417356 - Disruption of the gamma c cytokine network in t cells during hiv infection.
20210786 - Inflammatory cell recruitment in cardiovascular disease: murine models and potential cl...
Publication Detail:
Type:  Journal Article     Date:  2003-07-31
Journal Detail:
Title:  Blood     Volume:  102     ISSN:  0006-4971     ISO Abbreviation:  Blood     Publication Date:  2003 Nov 
Date Detail:
Created Date:  2003-11-04     Completed Date:  2004-01-05     Revised Date:  2004-11-17    
Medline Journal Info:
Nlm Unique ID:  7603509     Medline TA:  Blood     Country:  United States    
Other Details:
Languages:  eng     Pagination:  3719-26     Citation Subset:  AIM; IM    
Affiliation:
Laboratoire d'Immunopharmacologie, Institut National de la Santé et de la Recherche Médicale U503 and U404, Centre d'Etudes et de Recherche en Virologie et Immunologie, Lyon, France.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Antilymphocyte Serum / administration & dosage,  pharmacology*
Apoptosis*
Caspases
Cathepsin B / metabolism,  physiology*
Cytochromes c
Cytosol
Dose-Response Relationship, Drug
Humans
Lymphocyte Activation / drug effects
Lymphocyte Depletion / methods*
Lysosomes
T-Lymphocytes / cytology
Chemical
Reg. No./Substance:
0/Antilymphocyte Serum; 9007-43-6/Cytochromes c; EC 3.4.22.-/Caspases; EC 3.4.22.1/Cathepsin B

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Quantification of PRV-1 mRNA distinguishes polycythemia vera from secondary erythrocytosis.
Next Document:  Identification and characterization of 2 types of erythroid progenitors that express GATA-1 at disti...