Document Detail


Caspase-resistant vimentin suppresses apoptosis after photodynamic treatment with a silicon phthalocyanine in Jurkat cells.
MedLine Citation:
PMID:  11368515     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Oxidative stress, such as photodynamic therapy, is an apoptosis inducer. Apoptosis, as well as photosensitization, have been associated with disruption of the cytoskeletal network. The purpose of the present study was to assess the role of vimentin, a major cytoskeletal protein, in apoptosis after photodynamic treatment (PDT) with the silicon phthalocyanine Pc 4 in human Jurkat T cells. Here we show for the first time that photosensitization with Pc 4 initiates vimentin cleavage and that this event precedes poly(ADP-ribose) polymerase (PARP) degradation. Similar findings were obtained in the presence of C2-ceramide, an inducer of oxidative stress and apoptosis. In the presence of benzyloxycarbonyl-Val-Ala-Asp(O-methyl)-fluoromethylketone, a pan-caspase inhibitor, Pc 4-PDT-induced vimentin and PARP cleavage were abolished. In Jurkat cells transfected with a caspase-resistant vimentin apoptosis was partly suppressed and delayed post-Pc 4-PDT. We suggest that the full-length vimentin confers resistance to nuclear apoptosis after PDT with Pc 4.
Authors:
I Belichenko; N Morishima; D Separovic
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Archives of biochemistry and biophysics     Volume:  390     ISSN:  0003-9861     ISO Abbreviation:  Arch. Biochem. Biophys.     Publication Date:  2001 Jun 
Date Detail:
Created Date:  2001-05-22     Completed Date:  2001-06-28     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0372430     Medline TA:  Arch Biochem Biophys     Country:  United States    
Other Details:
Languages:  eng     Pagination:  57-63     Citation Subset:  IM    
Copyright Information:
Copyright 2001 Academic Press.
Affiliation:
Department of Radiation Oncology, Case Western Reserve University School of Medicine, Cleveland, Ohio, 44106-4942, USA.
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MeSH Terms
Descriptor/Qualifier:
Apoptosis / drug effects*
Caspases / pharmacology
Drug Resistance
Humans
Indoles / pharmacology*
Jurkat Cells
Organosilicon Compounds / pharmacology*
Oxidative Stress
Photochemotherapy
Photosensitizing Agents / pharmacology*
Poly(ADP-ribose) Polymerases / metabolism
Silanes*
Sphingosine / analogs & derivatives,  pharmacology
Transfection
Vimentin / genetics,  metabolism,  pharmacology*
Grant Support
ID/Acronym/Agency:
R29 CA77475/CA/NCI NIH HHS
Chemical
Reg. No./Substance:
0/Indoles; 0/N-acetylsphingosine; 0/Organosilicon Compounds; 0/Photosensitizing Agents; 0/Silanes; 0/Vimentin; 123-78-4/Sphingosine; 135719-28-7/silicon phthalocyanine; 92396-88-8/bis(tri-n-hexylsiloxy)(2,3-naphthalocyaninato)silicon; EC 2.4.2.30/Poly(ADP-ribose) Polymerases; EC 3.4.22.-/Caspases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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