Document Detail


Cardiogenic shock: the role of inflammation.
MedLine Citation:
PMID:  21039083     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Cardiogenic shock (CS) is the leading cause of death in patients with acute myocardial infarction (MI) and we badly need new approaches in its treatment. It has been demonstrated that a number of inflammatory cytokines (IL-1β, IL-6, IL-8, TNF-α, CRP, soluble adhesion molecules, complement system etc) are elevated in acute MI complicated by CS. Baseline levels of pro- inflammatory cytokines have predictive value for the development of CS and subsequent mortality. The deleterious effects of pro- inflammatory cytokines may be due to excessive nitric oxide production by enzyme named NOS. However in multicenter randomized TRIUMPH study non-selective NOS inhibition was ineffective in the treatment of cardiogenic shock. A challenging subject of future studies will be treatment of CS with specific inhibitors of inducible isoform of NOS. Considering the results of treatment of patients with septic shock it would be reasonable to study the effects of small doses of corticosteroids and hemofiltration in patients with CS and signs of SIRS.
Authors:
Alexander Shpektor
Related Documents :
10446833 - Microcirculatory oxygenation and shunting in sepsis and shock.
18086373 - Role of toll-like receptor responses for sepsis pathogenesis.
616743 - Interaction of intracellular proteases and immune mechanisms.
Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Acute cardiac care     Volume:  12     ISSN:  1748-295X     ISO Abbreviation:  Acute Card Care     Publication Date:  2010 Dec 
Date Detail:
Created Date:  2010-11-02     Completed Date:  2010-12-20     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101276603     Medline TA:  Acute Card Care     Country:  England    
Other Details:
Languages:  eng     Pagination:  115-8     Citation Subset:  IM    
Affiliation:
Cardiology Department, Moscow State University of Medicine and Dentistry, Moscow, Russia. moscowcardio_23@yahoo.com
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Adrenal Cortex Hormones / therapeutic use
Cytokines / physiology
Enzyme Inhibitors / therapeutic use
Hemofiltration
Humans
Myocardial Infarction / complications
Nitric Oxide / physiology
Nitric Oxide Synthase / antagonists & inhibitors,  physiology
Randomized Controlled Trials as Topic
Shock, Cardiogenic / etiology,  immunology*,  metabolism,  physiopathology,  therapy
Systemic Inflammatory Response Syndrome / immunology,  metabolism,  physiopathology*,  therapy
Treatment Outcome
Chemical
Reg. No./Substance:
0/Adrenal Cortex Hormones; 0/Cytokines; 0/Enzyme Inhibitors; 10102-43-9/Nitric Oxide; EC 1.14.13.39/Nitric Oxide Synthase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Taking a light approach to treating acute ischemic stroke patients: transcranial near-infrared laser...
Next Document:  Unprotected left main stenting, short- and long-term outcomes.