| Cardiac valve calcifications and left ventricular hypertrophy in hemodialysis patients. | |
| | |
MedLine Citation:
|
PMID: 16350826 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
|
Cardiac valve calcification (VC) is a common finding in end-stage renal disease patients. It was shown recently that VC is an independent predictor for all-cause and cardiovascular mortality in peritoneal dialysis patients. In hemodialysis (HD) patients, VC was associated with all-cause and cardiovascular mortality, but after adjusting for other cardiovascular risk factors and complications, as well as left ventricular mass index (LVMI), it lost significance. The aim of the study was to assess the relationship between VC and left ventricular hypertrophy in hemodialysis patients. Echocardiographic examination with mitral and aortic valves assessment and LVMI calculation was performed in 65 HD patients ages 49+/-12, with duration of HD therapy 38+/-32 months. VC were found in 32 of 65 patients (49%)-Group VC(+), mitral valve calcifications (MVC) in 10, aortic valve calcifications (AVC) in 9, and both valves calcifications (MVC+AVC) in 13 patients. Patients with VC were older, on HD therapy were longer, had higher systolic and pulse pressure, and had higher LVMI. Patients with both VCs had the highest LVMI. No significant differences were found with respect to Ca, P, PTH, and mean Ca x P product, but the incidence of Ca x P product above 4.43 mmol2/L2 was higher in VC(+) compared with those without VCs. VC coexists with left ventricular hypertrophy, particularly when both valves are calcified. Even short-lasting incidents of increased Ca x P product may lead to cardiac VC. |
| | |
Authors:
|
Pawe? Str?zecki; Grazyna Odrowaz-Sypniewska; Jacek Manitius |
Related Documents
:
|
2370256 - Surgery of type a acute aortic dissection with gelatine-resorcine-formol biological glu... 3272236 - Congenital left ventricular inflow obstruction: is the outcome related to the site of t... 8644676 - Comparison of infective endocarditis in patients with and without previously recognized... 17706566 - Fibrotic heart-valve reactions to dopamine-agonist treatment in parkinson's disease. 22578916 - Combination of noninvasive neurovascular imaging modalities in stroke patients: pattern... 2780446 - How do we know who has had deep vein thrombosis? |
Publication Detail:
|
Type: Comparative Study; Journal Article |
Journal Detail:
|
Title: Renal failure Volume: 27 ISSN: 0886-022X ISO Abbreviation: Ren Fail Publication Date: 2005 |
Date Detail:
|
Created Date: 2005-12-14 Completed Date: 2006-02-28 Revised Date: 2008-05-21 |
Medline Journal Info:
|
Nlm Unique ID: 8701128 Medline TA: Ren Fail Country: United States |
Other Details:
|
Languages: eng Pagination: 733-8 Citation Subset: IM |
Affiliation:
|
Department of Nephrology, Hypertension and Internal Diseases, Collegium Medicum in Bydgoszcz, Nicolaus Copernicus University, Toru?, Poland. st_pawel@cm.umk.pl |
Export Citation:
|
APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
|
Adult Age Distribution Aged Calcinosis / epidemiology, etiology*, ultrasonography Case-Control Studies Cohort Studies Echocardiography, Doppler Female Follow-Up Studies Heart Valve Diseases / epidemiology, etiology*, ultrasonography Humans Hypertrophy, Left Ventricular / epidemiology, etiology*, ultrasonography Incidence Kidney Failure, Chronic / diagnosis, therapy Male Middle Aged Poland / epidemiology Probability Renal Dialysis / adverse effects*, methods Risk Assessment Severity of Illness Index Sex Distribution Statistics, Nonparametric Time Factors |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
Previous Document: Effect of radiocontrasts on selected membrane transport systems.
Next Document: Relation of thyroid disfunction, thyroid autoantibodies, and renal function.