Document Detail


Cardiac uptake-1 inhibition by high circulating norepinephrine levels in patients with pheochromocytoma.
MedLine Citation:
PMID:  15078865     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Neuronal reuptake (uptake-1) constitutes the main route of inactivation of the sympathetic neurotransmitter norepinephrine in the heart and therefore contributes importantly to cardiac sympathetic neuroeffector function. In laboratory animals and in vitro preparations, half saturation of the transporter occurs at norepinephrine concentrations of 0.1 to 1 micromol/L. This study addressed whether endogenous norepinephrine can attain high enough plasma concentrations in humans to inhibit cardiac uptake-1. Patients with increased plasma norepinephrine levels due to pheochromocytoma were assessed by 6-[18F]fluorodopamine positron emission tomography. Above an antecubital venous plasma concentration of 3 nmol/L (approximately 500 pg/mL), left ventricular myocardial 6-[18F]fluorodopamine-derived radioactivity varied inversely with the logarithm of the plasma norepinephrine concentration (r=-0.77, P<0.0001). Reduction of plasma norepinephrine levels by treatment of the pheochromocytoma increased myocardial 6-[18F]fluorodopamine-derived radioactivity. At sufficiently high plasma concentrations, endogenous norepinephrine can compete with sympathetic imaging agents for uptake-1. The results call for caution in drawing quantitative conclusions about uptake-1 in the setting of high circulating concentrations of endogenous norepinephrine.
Authors:
Basil A Eldadah; Karel Pacak; Graeme Eisenhofer; Courtney Holmes; Irwin J Kopin; David S Goldstein
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Publication Detail:
Type:  Journal Article     Date:  2004-04-12
Journal Detail:
Title:  Hypertension     Volume:  43     ISSN:  1524-4563     ISO Abbreviation:  Hypertension     Publication Date:  2004 Jun 
Date Detail:
Created Date:  2004-05-28     Completed Date:  2004-10-28     Revised Date:  2005-11-17    
Medline Journal Info:
Nlm Unique ID:  7906255     Medline TA:  Hypertension     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1227-32     Citation Subset:  IM    
Affiliation:
Clinical Neurocardiology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, 10 Center Drive, MSC-1620, Bethesda, MD 20892-1620, USA. eldadahb@ninds.nih.gov
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MeSH Terms
Descriptor/Qualifier:
Adrenal Gland Neoplasms / metabolism*,  radionuclide imaging
Adult
Aged
Aged, 80 and over
Binding, Competitive
Dihydroxyphenylalanine / analogs & derivatives*,  diagnostic use,  pharmacokinetics
False Negative Reactions
Female
Heart / innervation,  radionuclide imaging*
Humans
Imaging, Three-Dimensional
Liver / metabolism
Male
Middle Aged
Myocardium / metabolism*
Norepinephrine / blood*
Norepinephrine Plasma Membrane Transport Proteins
Pheochromocytoma / metabolism*,  radionuclide imaging
Radiopharmaceuticals / diagnostic use,  pharmacokinetics
Symporters / antagonists & inhibitors*,  metabolism
Tomography, Emission-Computed*
Chemical
Reg. No./Substance:
0/Norepinephrine Plasma Membrane Transport Proteins; 0/Radiopharmaceuticals; 0/SLC6A2 protein, human; 0/Symporters; 51-41-2/Norepinephrine; 63-84-3/Dihydroxyphenylalanine; 75290-51-6/fluorodopa F 18

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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